Metabolomics and In-Silico Analysis Reveal Critical Energy Deregulations in Animal Models of Parkinson’s Disease

l-glutamine is a key parameter in the immunosuppression phenomenon

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Dynamic modelling of protein and oxidative metabolisms simulates the pathogenesis of Parkinson's disease

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Genetic etiology of Parkinson disease associated with mutations in the SNCA, PARK2, PINK1, PARK7, and LRRK2 genes: a mutation update

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BRENDA, the enzyme information system in 2011

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The control systems structures of energy metabolism

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PINK1-dependent recruitment of Parkin to mitochondria in mitophagy

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Computational analysis of determinants of dopamine (DA) dysfunction in DA nerve terminals

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Simultaneous extraction of several metabolites of energy metabolism and related substances in mammalian cells: Optimization using experimental design

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