Candidate Sequence Variants and Fetal Hemoglobin in Children with Sickle Cell Disease Treated with Hydroxyurea
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Title
Candidate Sequence Variants and Fetal Hemoglobin in Children with Sickle Cell Disease Treated with Hydroxyurea
Authors
Keywords
Genetic loci, Sickle cell disease, Molecular genetics, Hemoglobin, Globins, Children, Linear regression analysis, Pediatrics
Journal
PLoS One
Volume 8, Issue 2, Pages e55709
Publisher
Public Library of Science (PLoS)
Online
2013-02-08
DOI
10.1371/journal.pone.0055709
References
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Note: Only part of the references are listed.- Meta-analysis of 2040 sickle cell anemia patients: BCL11A and HBS1L-MYB are the major modifiers of HbF in African Americans
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- Fetal Hemoglobin Levels in African American and Hispanic Children With Sickle Cell Disease at Baseline and in Response to Hydroxyurea
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- Genetics of fetal hemoglobin in Tanzanian and British patients with sickle cell anemia
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- Control of fetal hemoglobin: new insights emerging from genomics and clinical implications
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- BCL11A is a major HbF quantitative trait locus in three different populations with β-hemoglobinopathies
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- Fetal haemoglobin response to hydroxycarbamide treatment and sar1a promoter polymorphisms in sickle cell anaemia
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- Hydroxyurea for the Treatment of Sickle Cell Anemia
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- DNA polymorphisms at the BCL11A, HBS1L-MYB, and -globin loci associate with fetal hemoglobin levels and pain crises in sickle cell disease
- (2008) G. Lettre et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Fetal hemoglobin in sickle cell anemia: Bayesian modeling of genetic associations
- (2007) Paola Sebastiani et al. AMERICAN JOURNAL OF HEMATOLOGY
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