In vivo key role of reactive oxygen species and NHE-1 activation in determining excessive cardiac hypertrophy

Linking Oxidative Stress, the Renin-Angiotensin System, and Hypertension

(2011) Camilla Fanelli et al.   HYPERTENSION

Regulation of myocardial growth and death by NADPH oxidase

(2011) Yasuhiro Maejima et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Role of autocrine/paracrine mechanisms in response to myocardial strain

(2011) Horacio E. Cingolani et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart

(2010) J. Kuroda et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Redox sensitive signaling pathways in cardiac remodeling, hypertrophy and failure

(2009) Narayana Anilkumar   Frontiers in Bioscience-Landmark

Losartan Metabolite EXP3179 Blocks NADPH Oxidase-Mediated Superoxide Production by Inhibiting Protein Kinase C

(2009) Ana Fortuño et al.   HYPERTENSION

Compensatory or inappropriate left ventricular mass in different models of left ventricular pressure overload: comparison between patients with aortic stenosis and arterial hypertension

(2009) Gian Francesco Mureddu et al.   JOURNAL OF HYPERTENSION

Chronic inhibition of the Na+/H+- exchanger causes regression of hypertrophy, heart failure, and ionic and electrophysiological remodelling

(2008) A Baartscheer et al.   BRITISH JOURNAL OF PHARMACOLOGY

Activation of Na + /H + Exchanger 1 Is Sufficient to Generate Ca 2+ Signals That Induce Cardiac Hypertrophy and Heart Failure

(2008) Tomoe Y. Nakamura et al.   CIRCULATION RESEARCH

Is cardiac hypertrophy a required compensatory mechanism in pressure-overloaded heart?

(2008) Jane-Lise Samuel et al.   JOURNAL OF HYPERTENSION

Physiological myocardial hypertrophy: how and why?

(2007) Daniele Catalucci   Frontiers in Bioscience-Landmark