T-helper 17 cells mediate the osteo/odontoclastogenesis induced by excessive orthodontic forces
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Title
T-helper 17 cells mediate the osteo/odontoclastogenesis induced by excessive orthodontic forces
Authors
Keywords
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Journal
ORAL DISEASES
Volume 18, Issue 4, Pages 375-388
Publisher
Wiley
Online
2011-12-06
DOI
10.1111/j.1601-0825.2011.01886.x
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Note: Only part of the references are listed.- IL-8 and MCP-1 induced by excessive orthodontic force mediates odontoclastogenesis in periodontal tissues
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- Peptidoglycan Enhances IL-6 Production in Human Synovial Fibroblasts via TLR2 Receptor, Focal Adhesion Kinase, Akt, and AP-1- Dependent Pathway
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- Interleukin-17 Promotes Autoimmunity by Triggering a Positive-Feedback Loop via Interleukin-6 Induction
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- Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infection
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- The differentiation of human TH-17 cells requires transforming growth factor-β and induction of the nuclear receptor RORγt
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- Low-energy laser stimulates tooth movement velocity via expression of RANK and RANKL
- (2008) S Fujita et al. Orthodontics & Craniofacial Research
- IL-6 trans-signalling directly induces RANKL on fibroblast-like synovial cells and is involved in RANKL induction by TNF- and IL-17
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- Evidence of the presence of T helper type 17 cells in chronic lesions of human periodontal disease
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- Effects of compression force on fibroblast growth factor-2 and receptor activator of nuclear factor kappa B ligand production by periodontal ligament cells in vitro
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