TDCPP protects cardiomyocytes from H2O2-induced injuries via activating PI3K/Akt/GSK3β signaling pathway
Published 2018 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
TDCPP protects cardiomyocytes from H2O2-induced injuries via activating PI3K/Akt/GSK3β signaling pathway
Authors
Keywords
TDCPP, Cardiomyocytes, Oxidative stress, Calcium overload, Apoptosis
Journal
MOLECULAR AND CELLULAR BIOCHEMISTRY
Volume -, Issue -, Pages -
Publisher
Springer Nature America, Inc
Online
2018-09-01
DOI
10.1007/s11010-018-3431-8
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- TDCPP protects cardiomyocytes from hypoxia-reoxygenation injury induced apoptosis through mitigating calcium overload and promotion GSK-3β phosphorylation
- (2018) Xiju He et al. REGULATORY TOXICOLOGY AND PHARMACOLOGY
- LncRNA CAIF inhibits autophagy and attenuates myocardial infarction by blocking p53-mediated myocardin transcription
- (2018) Cui-Yun Liu et al. Nature Communications
- Major contribution of the 3/6/7 class of TRPC channels to myocardial ischemia/reperfusion and cellular hypoxia/reoxygenation injuries
- (2017) Xiju He et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The mitochondrial permeability transition pore: Molecular nature and role as a target in cardioprotection
- (2015) Paolo Bernardi et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- 5-Methoxytryptophan-dependent protection of cardiomyocytes from heart ischemia reperfusion injury
- (2014) Hsiu-Chuan Chou et al. ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS
- Inhibition of Voltage-Gated Calcium Channels as Common Mode of Action for (Mixtures of) Distinct Classes of Insecticides
- (2014) Marieke Meijer et al. TOXICOLOGICAL SCIENCES
- Toxicity of TDCPP and TCEP on PC12 cell: Changes in CAMKII, GAP43, tubulin and NF-H gene and protein levels
- (2014) Na Ta et al. TOXICOLOGY LETTERS
- Resveratrol protects rabbit ventricular myocytes against oxidative stress-induced arrhythmogenic activity and Ca2+ overload
- (2013) Wei Li et al. ACTA PHARMACOLOGICA SINICA
- Historical Perspective on the Pathology of Myocardial Ischemia/Reperfusion Injury
- (2013) Robert B. Jennings CIRCULATION RESEARCH
- Modulation of cell viability, oxidative stress, calcium homeostasis, and voltage- and ligand-gated ion channels as common mechanisms of action of (mixtures of) non-dioxin-like polychlorinated biphenyls and polybrominated diphenyl ethers
- (2013) Remco H. S. Westerink ENVIRONMENTAL SCIENCE AND POLLUTION RESEARCH
- Exposure of zebrafish embryos/larvae to TDCPP alters concentrations of thyroid hormones and transcriptions of genes involved in the hypothalamic–pituitary–thyroid axis
- (2012) Qiangwei Wang et al. AQUATIC TOXICOLOGY
- Effects of tris(1,3-dichloro-2-propyl) phosphate and triphenyl phosphate on receptor-associated mRNA expression in zebrafish embryos/larvae
- (2012) Chunsheng Liu et al. AQUATIC TOXICOLOGY
- Hyaluronic acid-dependent protection in H9C2 cardiomyocytes: A cell model of heart ischemia–reperfusion injury and treatment
- (2012) Ching-Hsuan Law et al. TOXICOLOGY
- Reactive Oxygen Species–Activated Ca/Calmodulin Kinase IIδ Is Required for Late I Na Augmentation Leading to Cellular Na and Ca Overload
- (2011) Stefan Wagner et al. CIRCULATION RESEARCH
- Differential Roles of GSK-3β During Myocardial Ischemia and Ischemia/Reperfusion
- (2011) Peiyong Zhai et al. CIRCULATION RESEARCH
- Is the PentaBDE replacement, tris (1,3-dichloro-2-propyl) phosphate (TDCPP), a developmental neurotoxicant? Studies in PC12 cells
- (2011) Laura V. Dishaw et al. TOXICOLOGY AND APPLIED PHARMACOLOGY
- Oxidative stress caused by mitochondrial calcium overload
- (2010) Tsung-I Peng et al. Annals of the New York Academy of Sciences
- Cardioprotection by GSK-3 inhibition: role of enhanced glycogen synthesis and attenuation of calcium overload
- (2010) Mohamed A. Omar et al. CARDIOVASCULAR RESEARCH
- Prenatal Exposure to PBDEs and Neurodevelopment
- (2010) Julie B. Herbstman et al. ENVIRONMENTAL HEALTH PERSPECTIVES
- Proteomics study of oxidative stress and Src kinase inhibition in H9C2 cardiomyocytes: a cell model of heart ischemia–reperfusion injury and treatment
- (2010) Hsiu-Chuan Chou et al. FREE RADICAL BIOLOGY AND MEDICINE
- Reactive oxygen species in TNFα-induced signaling and cell death
- (2010) Michael J. Morgan et al. MOLECULES AND CELLS
- Calcium-Related Processes Involved in the Inhibition of Depolarization-Evoked Calcium Increase by Hydroxylated PBDEs in PC12 Cells
- (2010) Milou M. L. Dingemans et al. TOXICOLOGICAL SCIENCES
- GSK-3β, a Therapeutic Target for Cardiomyocyte Protection
- (2009) Tetsuji Miura et al. CIRCULATION JOURNAL
- Role of Glycogen Synthase Kinase-3β in Cardioprotection
- (2009) Magdalena Juhaszova et al. CIRCULATION RESEARCH
- Bromination Pattern of Hydroxylated Metabolites of BDE-47 Affects Their Potency to Release Calcium from Intracellular Stores in PC12 Cells
- (2009) Milou M.L. Dingemans et al. ENVIRONMENTAL HEALTH PERSPECTIVES
- House Dust Concentrations of Organophosphate Flame Retardants in Relation to Hormone Levels and Semen Quality Parameters
- (2009) John D. Meeker et al. ENVIRONMENTAL HEALTH PERSPECTIVES
- Polybrominated diphenyl ether (PBDE) concentrations in house dust are related to hormone levels in men
- (2009) John D. Meeker et al. SCIENCE OF THE TOTAL ENVIRONMENT
- Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling
- (2008) G. W. Dorn CARDIOVASCULAR RESEARCH
- Hydroxylation Increases the Neurotoxic Potential of BDE-47 to Affect Exocytosis and Calcium Homeostasis in PC12 Cells
- (2008) Milou M.L. Dingemans et al. ENVIRONMENTAL HEALTH PERSPECTIVES
- Mechanisms Underlying Acute Protection From Cardiac Ischemia-Reperfusion Injury
- (2008) Elizabeth Murphy et al. PHYSIOLOGICAL REVIEWS
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started