ZEB1 overexpression associated with E-cadherin and microRNA-200 downregulation is characteristic of undifferentiated endometrial carcinoma

RASSF1A and CDH1 hypermethylation as potential epimarkers in breast cancer

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Molecular Profile of Grade 3 Endometrioid Endometrial Carcinoma

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HMGA2 is commonly expressed in uterine serous carcinomas and is a useful adjunct to diagnosis

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Molecular events in endometrial carcinosarcomas and the role of high mobility group AT-hook 2 in endometrial carcinogenesis

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Two Cases of Low-Grade Endometriod Carcinoma Associated with Undifferentiated Carcinoma of the Uterus (Dedifferentiated Carcinoma): A Molecular Study

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The emerging role of miR-200 family of MicroRNAs in epithelial-mesenchymal transition and cancer metastasis

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Steroid receptors and microRNAs: Relationships revealed

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DNA Mismatch Repair Deficiency in Endometrial Carcinoma

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The EMT-activator ZEB1 promotes tumorigenicity by repressing stemness-inhibiting microRNAs

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Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like Phenotype

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A Double-Negative Feedback Loop between ZEB1-SIP1 and the microRNA-200 Family Regulates Epithelial-Mesenchymal Transition

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

(2008) Ulrike Burk et al.   EMBO REPORTS

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

(2008) S.-M. Park et al.   GENES & DEVELOPMENT

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(2008) Meenakshi Singh et al.   MODERN PATHOLOGY

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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