STAT3 Tyr705 phosphorylation affects clinical outcome in patients with newly diagnosed supratentorial glioblastoma
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Title
STAT3 Tyr705 phosphorylation affects clinical outcome in patients with newly diagnosed supratentorial glioblastoma
Authors
Keywords
Glioblastoma, STAT3, Tyrosine, Phosphorylation, Survival
Journal
MEDICAL ONCOLOGY
Volume 31, Issue 4, Pages -
Publisher
Springer Nature
Online
2014-03-20
DOI
10.1007/s12032-014-0924-5
References
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- Targeting Role of Glioma Stem Cells for Gliobastoma Multiforme
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- Migfilin Protein Promotes Migration and Invasion in Human Glioma through Epidermal Growth Factor Receptor-mediated Phospholipase C-γ and STAT3 Protein Signaling Pathways
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- STAT3 tyrosine phosphorylation influences survival in glioblastoma
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- Inhibition of the JAK-2/STAT3 signaling pathway impedes the migratory and invasive potential of human glioblastoma cells
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- STAT3 Regulation of Glioblastoma Pathogenesis
- (2009) Nuria la Iglesia et al. CURRENT MOLECULAR MEDICINE
- Stat3 activation is required for the growth of U87 cell-derived tumours in mice
- (2009) Atreyi Dasgupta et al. EUROPEAN JOURNAL OF CANCER
- STAT3 Is Required for Proliferation and Maintenance of Multipotency in Glioblastoma Stem Cells
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- The Incidence, Correlation with Tumor-Infiltrating Inflammation, and Prognosis of Phosphorylated STAT3 Expression in Human Gliomas
- (2008) M. Abou-Ghazal et al. CLINICAL CANCER RESEARCH
- Signal Transducer and Activator of Transcription-3: A Molecular Hub for Signaling Pathways in Gliomas
- (2008) E. C. Brantley et al. MOLECULAR CANCER RESEARCH
- STAT3: A critical transcription activator in angiogenesis
- (2007) Zhong Chen et al. MEDICINAL RESEARCH REVIEWS
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