Cell-specific effects of TNF-α and IL-1β on alkaline phosphatase: implication for syndesmophyte formation and vascular calcification

Physiological role of alkaline phosphatase explored in hypophosphatasia

(2010) Michael P. Whyte   Annals of the New York Academy of Sciences

Can we stop progression of ankylosing spondylitis?

(2010) Georg Schett et al.   BEST PRACTICE & RESEARCH IN CLINICAL RHEUMATOLOGY

TNF-α stimulates alkaline phosphatase and mineralization through PPARγ inhibition in human osteoblasts

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Do cytokines induce vascular calcification by the mere stimulation of TNAP activity?

(2010) P. Lencel et al.   MEDICAL HYPOTHESES

Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci

(2010) John D Reveille et al.   NATURE GENETICS

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(2010) Lai-Shan Tam et al.   Nature Reviews Rheumatology

Differential effects of hypoxia on osteochondrogenic potential of human adipose-derived stem cells

(2009) Christophe Merceron et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Inflammatory lesions of the spine on magnetic resonance imaging predict the development of new syndesmophytes in ankylosing spondylitis: Evidence of a relationship between inflammation and new bone formation

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Bone morphogenetic protein signaling and arthritis

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TNF-α and IL-1β inhibit RUNX2 and collagen expression but increase alkaline phosphatase activity and mineralization in human mesenchymal stem cells

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Evidence for a distinctive pattern of bone formation in enthesophytes

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Regulation of PPARγ function by TNF-α

(2008) Jianping Ye   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Exuberant expression of chemokine genes by adult human articular chondrocytes in response to IL-1β

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The relationship between inflammation and new bone formation in patients with ankylosing spondylitis

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