Infection of Vascular Endothelial Cells with Human Cytomegalovirus under Fluid Shear Stress Reveals Preferential Entry and Spread of Virus in Flow Conditions Simulating Atheroprone Regions of the Artery

Key transcriptional regulators of the vasoprotective effects of shear stress

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Cytomegalovirus-Induced Effector T Cells Cause Endothelial Cell Damage

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Human Cytomegalovirus Induces Multiple Means To Combat Reactive Oxygen Species

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Effects of Disturbed Flow on Vascular Endothelium: Pathophysiological Basis and Clinical Perspectives

(2011) Jeng-Jiann Chiu et al.   PHYSIOLOGICAL REVIEWS

Cytomegalovirus Seroprevalence in the United States: The National Health and Nutrition Examination Surveys, 1988–2004

(2010) Sheri Lewis Bate et al.   CLINICAL INFECTIOUS DISEASES

The Activator Protein 1 Binding Motifs within the Human Cytomegalovirus Major Immediate-Early Enhancer Are Functionally Redundant and Act in a Cooperative Manner with the NF- B Sites during Acute Infection

(2010) E. Isern et al.   JOURNAL OF VIROLOGY

Endothelial cell responses to atheroprone flow are driven by two separate flow components: low time-average shear stress and fluid flow reversal

(2009) Daniel E. Conway et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Cytomegalovirus Infection Causes an Increase of Arterial Blood Pressure

(2009) Jilin Cheng et al.   PLoS Pathogens

Inflammation at the Molecular Interface of Atherogenesis

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Detection of Human Cytomegalovirus in Atherosclerotic Carotid Arteries in Humans

(2008) Li Yi et al.   JOURNAL OF THE FORMOSAN MEDICAL ASSOCIATION

Human Cytomegalovirus Secretome Contains Factors That Induce Angiogenesis and Wound Healing

(2008) J. Dumortier et al.   JOURNAL OF VIROLOGY

Platelet-derived growth factor-α receptor activation is required for human cytomegalovirus infection

(2008) Liliana Soroceanu et al.   NATURE

Human CMV infection of endothelial cells induces an angiogenic response through viral binding to EGF receptor and  1 and  3 integrins

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