A Functional Role for TorsinA in Herpes Simplex Virus 1 Nuclear Egress
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Title
A Functional Role for TorsinA in Herpes Simplex Virus 1 Nuclear Egress
Authors
Keywords
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Journal
JOURNAL OF VIROLOGY
Volume 85, Issue 19, Pages 9667-9679
Publisher
American Society for Microbiology
Online
2011-07-21
DOI
10.1128/jvi.05314-11
References
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Related references
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- A molecular mechanism underlying the neural-specific defect in torsinA mutant mice
- (2010) C. E. Kim et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Diseases of the Nuclear Envelope
- (2010) H. J. Worman et al. Cold Spring Harbor Perspectives in Biology
- Relative tissue expression of homologous torsinB correlates with the neuronal specific importance of DYT1 dystonia-associated torsinA
- (2009) M. Jungwirth et al. HUMAN MOLECULAR GENETICS
- TorsinA and dystonia: from nuclear envelope to synapse
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- LULL1 Retargets TorsinA to the Nuclear Envelope Revealing an Activity That Is Impaired by the DYT1 Dystonia Mutation
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- Transcriptional and proteomic profiling in a cellular model of DYT1 dystonia
- (2009) J.N. Martin et al. NEUROSCIENCE
- Herpesvirus assembly: An update
- (2009) Thomas C. Mettenleiter et al. VIRUS RESEARCH
- siRNA knock-down of mutant torsinA restores processing through secretory pathway in DYT1 dystonia cells
- (2008) J. W. Hewett et al. HUMAN MOLECULAR GENETICS
- Dystonia-associated mutations cause premature degradation of torsinA protein and cell-type-specific mislocalization to the nuclear envelope
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