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Reduced carbohydrate availability enhances exercise-induced p53 signaling in human skeletal muscle: implications for mitochondrial biogenesis

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p53 Orchestrates the PGC-1α-Mediated Antioxidant Response Upon Mild Redox and Metabolic Imbalance

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Matched work high-intensity interval and continuous running induce similar increases in PGC-1α mRNA, AMPK, p38, and p53 phosphorylation in human skeletal muscle

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Sirtuin 1 (SIRT1) Deacetylase Activity Is Not Required for Mitochondrial Biogenesis or Peroxisome Proliferator-activated Receptor-γ Coactivator-1α (PGC-1α) Deacetylation following Endurance Exercise

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Exercise Increases Mitochondrial PGC-1α Content and Promotes Nuclear-Mitochondrial Cross-talk to Coordinate Mitochondrial Biogenesis

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A systematic review of the evidence for Canada's Physical Activity Guidelines for Adults

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Muscle-specific differences in the response of mitochondrial proteins to β-GPA feeding: an evaluation of potential mechanisms

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Specific attenuation of protein kinase phosphorylation in muscle with a high mitochondrial content

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Loss of p53 causes mitochondrial DNA depletion and altered mitochondrial reactive oxygen species homeostasis

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p53 Improves Aerobic Exercise Capacity and Augments Skeletal Muscle Mitochondrial DNA Content

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Biophysical characterizations of human mitochondrial transcription factor A and its binding to tumor suppressor p53

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Bimodal Protein Targeting through Activation of Cryptic Mitochondrial Targeting Signals by an Inducible Cytosolic Endoprotease

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Role of p53 in mitochondrial biogenesis and apoptosis in skeletal muscle

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High-fat diets cause insulin resistance despite an increase in muscle mitochondria

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