Intraneuronal Immunoreactivity for the Prion Protein Distinguishes a Subset of E200K Genetic From Sporadic Creutzfeldt-Jakob Disease

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Cyclosporin-A-induced prion protein aggresomes are dynamic quality-control cellular compartments

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Autophagy Contributes to Widespread Neuronal Degeneration in Hamsters Infected with the Echigo-1 Strain of Creutzfeldt-Jakob Disease and Mice Infected with the Fujisaki Strain of Gerstmann-Sträussler-Scheinker (GSS) Syndrome

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Fatal Prion Disease in a Mouse Model of Genetic E200K Creutzfeldt-Jakob Disease

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Cellular and sub-cellular pathology of animal prion diseases: relationship between morphological changes, accumulation of abnormal prion protein and clinical disease

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Genetic Creutzfeldt-Jakob disease associated with the E200K mutation: characterization of a complex proteinopathy

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Constant-pH Molecular Dynamics Simulations Reveal a β-Rich Form of the Human Prion Protein

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Fragment molecular orbital calculations reveal that the E200K mutation markedly alters local structural stability in the human prion protein

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Similarities between Forms of Sheep Scrapie and Creutzfeldt-Jakob Disease Are Encoded by Distinct Prion Types

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Autophagy induction by trehalose counter-acts cellular prion-infection

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Lithium induces clearance of protease resistant prion protein in prion-infected cells by induction of autophagy

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Prion protein biosynthesis and its emerging role in neurodegeneration

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Identification of an Intracellular Site of Prion Conversion

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