Primary phagocytosis of viable neurons by microglia activated with LPS or Aβ is dependent on calreticulin/LRP phagocytic signalling
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Title
Primary phagocytosis of viable neurons by microglia activated with LPS or Aβ is dependent on calreticulin/LRP phagocytic signalling
Authors
Keywords
-
Journal
Journal of Neuroinflammation
Volume 9, Issue 1, Pages -
Publisher
Springer Nature
Online
2012-08-14
DOI
10.1186/1742-2094-9-196
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Note: Only part of the references are listed.- A novel pathway combining calreticulin exposure and ATP secretion in immunogenic cancer cell death
- (2012) Abhishek D Garg et al. EMBO JOURNAL
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- Eaten alive! Cell death by primary phagocytosis: ‘phagoptosis’
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- (2011) Kodi S. Ravichandran IMMUNITY
- Neuronal Death Induced by Nanomolar Amyloid β Is Mediated by Primary Phagocytosis of Neurons by Microglia
- (2011) Urte Neniskyte et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Inhibition of Microglial Phagocytosis Is Sufficient To Prevent Inflammatory Neuronal Death
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- Caspase signalling controls microglia activation and neurotoxicity
- (2011) Miguel A. Burguillos et al. NATURE
- Physiology of Microglia
- (2011) Helmut Kettenmann et al. PHYSIOLOGICAL REVIEWS
- Surface-exposed calreticulin in the interaction between dying cells and phagocytes
- (2010) Isabelle Martins et al. Annals of the New York Academy of Sciences
- A Mechanism of Release of Calreticulin from Cells During Apoptosis
- (2010) Joanna M. Tarr et al. JOURNAL OF MOLECULAR BIOLOGY
- Inflammatory Neurodegeneration and Mechanisms of Microglial Killing of Neurons
- (2010) Guy C. Brown et al. MOLECULAR NEUROBIOLOGY
- CD47 Is Upregulated on Circulating Hematopoietic Stem Cells and Leukemia Cells to Avoid Phagocytosis
- (2009) Siddhartha Jaiswal et al. CELL
- Induction of caspase- and reactive oxygen species-independent phosphatidylserine externalization in primary human neutrophils: role in macrophage recognition and engulfment
- (2008) Siriporn Jitkaew et al. JOURNAL OF LEUKOCYTE BIOLOGY
- PAI-1 inhibits neutrophil efferocytosis
- (2008) Y.-J. Park et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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