The Mycobacterium tuberculosis Stress Response Factor SigH Is Required for Bacterial Burden as Well as Immunopathology in Primate Lungs
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
The Mycobacterium tuberculosis Stress Response Factor SigH Is Required for Bacterial Burden as Well as Immunopathology in Primate Lungs
Authors
Keywords
-
Journal
JOURNAL OF INFECTIOUS DISEASES
Volume 205, Issue 8, Pages 1203-1213
Publisher
Oxford University Press (OUP)
Online
2012-03-08
DOI
10.1093/infdis/jis102
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Foxp3-positive macrophages display immunosuppressive properties and promote tumor growth
- (2011) Soraya Zorro Manrique et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Reactivation of latent tuberculosis in rhesus macaques by coinfection with simian immunodeficiency virus
- (2011) Smriti Mehra et al. JOURNAL OF MEDICAL PRIMATOLOGY
- Decrease in the Effectiveness of Bacille Calmette‐Guérin Vaccine against Pulmonary Tuberculosis: A Consequence of Increased Immune Suppression by Microbial Antioxidants, Not Overattenuation
- (2010) Douglas S. Kernodle CLINICAL INFECTIOUS DISEASES
- Immunogenicity and Protection Induced by a Mycobacterium tuberculosis sigE Mutant in a BALB/c Mouse Model of Progressive Pulmonary Tuberculosis
- (2010) R. H. Pando et al. INFECTION AND IMMUNITY
- Exogenous Nef Is an Inhibitor ofMycobacterium tuberculosis-induced Tumor Necrosis Factor-α Production and Macrophage Apoptosis
- (2010) Kuldeep Kumawat et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Limited Role for Lymphotoxin in the Host Immune Response to Mycobacterium tuberculosis
- (2010) N. Allie et al. JOURNAL OF IMMUNOLOGY
- Mycobacterium tuberculosisMT2816 Encodes a Key Stress‐Response Regulator
- (2010) Smriti Mehra et al. JOURNAL OF INFECTIOUS DISEASES
- Genetic Requirements for the Survival of Tubercle Bacilli in Primates
- (2010) Noton K. Dutta et al. JOURNAL OF INFECTIOUS DISEASES
- CD4+Regulatory T Cells in a Cynomolgus Macaque Model ofMycobacterium tuberculosisInfection
- (2010) Angela M. Green et al. JOURNAL OF INFECTIOUS DISEASES
- A Mycobacterium tuberculosis Sigma Factor Network Responds to Cell-Envelope Damage by the Promising Anti-Mycobacterial Thioridazine
- (2010) Noton K. Dutta et al. PLoS One
- Transcriptional Reprogramming in Nonhuman Primate (Rhesus Macaque) Tuberculosis Granulomas
- (2010) Smriti Mehra et al. PLoS One
- Characterization of a Clp Protease Gene Regulator and the Reaeration Response in Mycobacterium tuberculosis
- (2010) Ashley M. Sherrid et al. PLoS One
- The level of monocyte turnover predicts disease progression in the macaque model of AIDS
- (2009) A. Hasegawa et al. BLOOD
- Functional Genomics Reveals Extended Roles of the Mycobacterium tuberculosis Stress Response Factor H
- (2009) S. Mehra et al. JOURNAL OF BACTERIOLOGY
- The Mycobacterium tuberculosis Sigma Factor B Is Required for Full Response to Cell Envelope Stress and Hypoxia In Vitro, but It Is Dispensable for In Vivo Growth
- (2009) P. A. Fontan et al. JOURNAL OF BACTERIOLOGY
- Global transcriptional response to vancomycin in Mycobacterium tuberculosis
- (2009) R. Provvedi et al. MICROBIOLOGY-SGM
- A replication clock for Mycobacterium tuberculosis
- (2009) Wendy P Gill et al. NATURE MEDICINE
- Reducing the Activity and Secretion of Microbial Antioxidants Enhances the Immunogenicity of BCG
- (2009) Shanmugalakshmi Sadagopal et al. PLoS One
- Tuberculous Granuloma Induction via Interaction of a Bacterial Secreted Protein with Host Epithelium
- (2009) H. E. Volkman et al. SCIENCE
- Mycobacterium tuberculosisSigma Factor E Regulon Modulates the Host Inflammatory Response
- (2008) Patricia A. Fontán et al. JOURNAL OF INFECTIOUS DISEASES
- Defective tryptophan catabolism underlies inflammation in mouse chronic granulomatous disease
- (2008) Luigina Romani et al. NATURE
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started