A study of molecular changes relating to energy metabolism and cellular stress in people with Huntington’s disease: looking for biomarkers

Validation of Plasma Branched Chain Amino Acids as Biomarkers in Huntington Disease

(2011) Fanny Mochel et al.   ARCHIVES OF NEUROLOGY

Mutant huntingtin causes defective actin remodeling during stress: defining a new role for transglutaminase 2 in neurodegenerative disease

(2011) Lise Munsie et al.   HUMAN MOLECULAR GENETICS

Early Alterations of Brain Cellular Energy Homeostasis in Huntington Disease Models

(2011) Fanny Mochel et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Brain-Derived Neurotrophic Factor in Patients with Huntington's Disease

(2011) Chiara Zuccato et al.   PLoS One

Up-regulation of the isoenzymes MAO-A and MAO-B in the human basal ganglia and pons in Huntington's disease revealed by quantitative enzyme radioautography

(2010) Grayson Richards et al.   BRAIN RESEARCH

Safinamide in the treatment of Parkinson's disease

(2010) Anthony HV Schapira   EXPERT OPINION ON PHARMACOTHERAPY

Impairment of PGC-1alpha expression, neuropathology and hepatic steatosis in a transgenic mouse model of Huntington's disease following chronic energy deprivation

(2010) R. K. Chaturvedi et al.   HUMAN MOLECULAR GENETICS

Huntington's disease: from molecular pathogenesis to clinical treatment

(2010) Christopher A Ross et al.   LANCET NEUROLOGY

Trinucleotide repeats: triggers for genomic disorders?

(2010) Piotr Kozlowski et al.   Genome Medicine

Functional mechanism of neuroprotection by inhibitors of type B monoamine oxidase in Parkinson’s disease

(2009) Makoto Naoi et al.   Expert Review of Neurotherapeutics

Polymorphisms in SLC6A4, PAH, GABRB3, and MAOB and modification of psychotic disorder features

(2009) Sarah E. Bergen et al.   SCHIZOPHRENIA RESEARCH

A novel pathogenic pathway of immune activation detectable before clinical onset in Huntington's disease

(2008) Maria Björkqvist et al.   JOURNAL OF EXPERIMENTAL MEDICINE

Pathogenesis of Inclusion Bodies in (CAG)n/Qn-Expansion Diseases with Special Reference to the Role of Tissue Transglutaminase and to Selective Vulnerability

(2003) Arthur J. L. Cooper et al.   JOURNAL OF NEUROCHEMISTRY