Association of nod-like receptor protein-3 single nucleotide gene polymorphisms and expression with the susceptibility to relapsing-remitting multiple sclerosis
Published 2018 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Association of nod-like receptor protein-3 single nucleotide gene polymorphisms and expression with the susceptibility to relapsing-remitting multiple sclerosis
Authors
Keywords
-
Journal
International Journal of Immunogenetics
Volume -, Issue -, Pages -
Publisher
Wiley
Online
2018-09-28
DOI
10.1111/iji.12401
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- The Effects of IFN-β 1a on the Expression of Inflammasomes and Apoptosis-Associated Speck-Like Proteins in Multiple Sclerosis Patients
- (2016) Saam Noroozi et al. MOLECULAR NEUROBIOLOGY
- Multiple Sclerosis Epidemiology in East Asia, South East Asia and South Asia: A Systematic Review
- (2016) Sharareh Eskandarieh et al. NEUROEPIDEMIOLOGY
- Differential Frequency of CD8+ T Cell Subsets in Multiple Sclerosis Patients with Various Clinical Patterns
- (2016) Zahra Salehi et al. PLoS One
- NLRP3 inflammasome is associated with the response to IFN-β in patients with multiple sclerosis
- (2015) Sunny Malhotra et al. BRAIN
- NLRP3gene polymorphisms in Iranian patients with recurrent aphthous stomatitis
- (2015) Alireza Zare Bidoki et al. JOURNAL OF ORAL PATHOLOGY & MEDICINE
- Role of Genetic Alterations in theNLRP3andCARD8Genes in Health and Disease
- (2015) G. V. Paramel et al. MEDIATORS OF INFLAMMATION
- Increased inflammasome related gene expression profile in PBMC may facilitate T helper 17 cell induction in multiple sclerosis
- (2015) E. Peelen et al. MOLECULAR IMMUNOLOGY
- Inflammasomes: mechanism of action, role in disease and therapeutics
- (2015) Haitao Guo et al. NATURE MEDICINE
- NLRP3Inflammasome Polymorphism and Macrovascular Complications in Type 2 Diabetes Patients
- (2015) Jasna Klen et al. Journal of Diabetes Research
- Estimated Prevalence and Incidence of Multiple Sclerosis in Iran
- (2014) Masoud Etemadifar et al. EUROPEAN NEUROLOGY
- 25-Hydroxycholesterol suppresses interleukin-1-driven inflammation downstream of type I interferon
- (2014) A. Reboldi et al. SCIENCE
- Evidence of NLRP3-inflammasome activation in rheumatoid arthritis (RA); genetic variants within the NLRP3-inflammasome complex in relation to susceptibility to RA and response to anti-TNF treatment
- (2013) Rebeccah J Mathews et al. ANNALS OF THE RHEUMATIC DISEASES
- Upregulated NLRP3 Inflammasome Activation in Patients With Type 2 Diabetes
- (2012) Hye-Mi Lee et al. DIABETES
- NLRP3 is activated in Alzheimer’s disease and contributes to pathology in APP/PS1 mice
- (2012) Michael T. Heneka et al. NATURE
- The Q705K Polymorphism in NLRP3 Is a Gain-of-Function Alteration Leading to Excessive Interleukin-1β and IL-18 Production
- (2012) Deepti Verma et al. PLoS One
- Activated Human CD4+CD45RO+ Memory T-Cells Indirectly Inhibit NLRP3 Inflammasome Activation through Downregulation of P2X7R Signalling
- (2012) Vanessa Beynon et al. PLoS One
- NLRP3 inflammasome induces chemotactic immune cell migration to the CNS in experimental autoimmune encephalomyelitis
- (2012) M. Inoue et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Diagnostic criteria for multiple sclerosis: 2010 Revisions to the McDonald criteria
- (2011) Chris H. Polman et al. ANNALS OF NEUROLOGY
- Interleukin-1β causes synaptic hyperexcitability in multiple sclerosis
- (2011) Silvia Rossi et al. ANNALS OF NEUROLOGY
- Understanding the mechanism of IL-1β secretion
- (2011) Gloria Lopez-Castejon et al. CYTOKINE & GROWTH FACTOR REVIEWS
- Type I Interferon Inhibits Interleukin-1 Production and Inflammasome Activation
- (2011) Greta Guarda et al. IMMUNITY
- Cytokines Genes Polymorphisms and Risk of Multiple Sclerosis
- (2010) Maryam Izad et al. AMERICAN JOURNAL OF THE MEDICAL SCIENCES
- The Inflammasomes
- (2010) Kate Schroder et al. CELL
- Evidence of interaction of CARD8 rs2043211 with NALP3 rs35829419 in Crohn's disease
- (2010) R L Roberts et al. GENES AND IMMUNITY
- A 3′UTR SNP in NLRP3 Gene is Associated With Susceptibility to HIV-1 Infection
- (2010) Alessandra Pontillo et al. JAIDS-JOURNAL OF ACQUIRED IMMUNE DEFICIENCY SYNDROMES
- Inflammation in the Central Nervous System and Th17 Responses Are Inhibited by IFN- -Induced IL-18 Binding Protein
- (2010) J. M. Millward et al. JOURNAL OF IMMUNOLOGY
- NLRP3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th1 and Th17 Responses
- (2010) D. Gris et al. JOURNAL OF IMMUNOLOGY
- The Inflammasome Sensor, NLRP3, Regulates CNS Inflammation and Demyelination via Caspase-1 and Interleukin-18
- (2010) S. Jha et al. JOURNAL OF NEUROSCIENCE
- Associations of functional NLRP3 polymorphisms with susceptibility to food-induced anaphylaxis and aspirin-induced asthma
- (2009) Yuki Hitomi et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- Role of the innate immune system in the pathogenesis of multiple sclerosis
- (2009) Roopali Gandhi et al. JOURNAL OF NEUROIMMUNOLOGY
- Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence
- (2009) Olaf Gross et al. NATURE
- Autoinflammatory genes and susceptibility to psoriatic juvenile idiopathic arthritis
- (2008) T. G. Day et al. ARTHRITIS AND RHEUMATISM
- A shift from adaptive to innate immunity: a potential mechanism of disease progression in multiple sclerosis
- (2008) Howard L. Weiner JOURNAL OF NEUROLOGY
- Differential diagnosis of suspected multiple sclerosis: a consensus approach
- (2008) DH Miller et al. Multiple Sclerosis Journal
- Common variants in the NLRP3 region contribute to Crohn's disease susceptibility
- (2008) Alexandra-Chloé Villani et al. NATURE GENETICS
- Interleukin-17 Production in Central Nervous System-Infiltrating T Cells and Glial Cells Is Associated with Active Disease in Multiple Sclerosis
- (2007) John S. Tzartos et al. AMERICAN JOURNAL OF PATHOLOGY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started