C5a2 can modulate ERK1/2 signaling in macrophages via heteromer formation with C5a1 and β-arrestin recruitment
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Title
C5a2 can modulate ERK1/2 signaling in macrophages via heteromer formation with C5a1 and β-arrestin recruitment
Authors
Keywords
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Journal
IMMUNOLOGY AND CELL BIOLOGY
Volume 92, Issue 7, Pages 631-639
Publisher
Wiley
Online
2014-04-29
DOI
10.1038/icb.2014.32
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Note: Only part of the references are listed.- The interaction between C5a and both C5aR and C5L2 receptors is required for production of G-CSF during acute inflammation
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- (2013) Daniel E Croker et al. IMMUNOLOGY AND CELL BIOLOGY
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- (2013) R. Wang et al. JOURNAL OF IMMUNOLOGY
- Human colonic epithelial cells detect and respond to C5a via apically expressed C5aR through the ERK pathway
- (2012) Qi Cao et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- C5L2 and C5aR interaction in adipocytes and macrophages: Insights into adipoimmunology
- (2012) Pegah Poursharifi et al. CELLULAR SIGNALLING
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- (2012) Rui Li et al. FASEB JOURNAL
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- G protein-coupled receptors: walking hand-in-hand, talking hand-in-hand?
- (2011) Henry F Vischer et al. BRITISH JOURNAL OF PHARMACOLOGY
- Beta-arrestin 2 negatively regulates sepsis-induced inflammation
- (2010) Hongkuan Fan et al. IMMUNOLOGY
- The C5a Receptor (C5aR) C5L2 Is a Modulator of C5aR-mediated Signal Transduction
- (2010) Claire E. Bamberg et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Time-resolved FRET between GPCR ligands reveals oligomers in native tissues
- (2010) Laura Albizu et al. Nature Chemical Biology
- C3a and C5a Are Chemotactic Factors for Human Mesenchymal Stem Cells, Which Cause Prolonged ERK1/2 Phosphorylation
- (2009) I. U. Schraufstatter et al. JOURNAL OF IMMUNOLOGY
- The human complement fragment receptor, C5L2, is a recycling decoy receptor
- (2008) Anne-Marie Scola et al. MOLECULAR IMMUNOLOGY
- Functional roles for C5a receptors in sepsis
- (2008) Daniel Rittirsch et al. NATURE MEDICINE
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