MicroRNA-146a and RBM4 form a negative feed-forward loop that disrupts cytokine mRNA translation following TLR4 responses in human THP-1 monocytes
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
MicroRNA-146a and RBM4 form a negative feed-forward loop that disrupts cytokine mRNA translation following TLR4 responses in human THP-1 monocytes
Authors
Keywords
-
Journal
IMMUNOLOGY AND CELL BIOLOGY
Volume 91, Issue 8, Pages 532-540
Publisher
Wiley
Online
2013-07-30
DOI
10.1038/icb.2013.37
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Adoptive transfer of CD34+ cells during murine sepsis rebalances macrophage lipopolysaccharide responses
- (2012) Laura Brudecki et al. IMMUNOLOGY AND CELL BIOLOGY
- Epigenetics, bioenergetics, and microRNA coordinate gene-specific reprogramming during acute systemic inflammation
- (2011) Charles E. McCall et al. JOURNAL OF LEUKOCYTE BIOLOGY
- MicroRNA-146a regulates both transcription silencing and translation disruption of TNF-α during TLR4-induced gene reprogramming
- (2011) Mohamed El Gazzar et al. JOURNAL OF LEUKOCYTE BIOLOGY
- MicroRNAs Distinguish Translational from Transcriptional Silencing during Endotoxin Tolerance
- (2010) Mohamed El Gazzar et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Gene-Specific Epigenetic Regulation in Serious Infections with Systemic Inflammation
- (2010) Charles E. McCall et al. Journal of Innate Immunity
- Development of endotoxin tolerance in humans in vivo
- (2009) Annelies Draisma et al. CRITICAL CARE MEDICINE
- Cooperative NCoR/SMRT interactions establish a corepressor-based strategy for integration of inflammatory and anti-inflammatory signaling pathways
- (2009) S. Ghisletti et al. GENES & DEVELOPMENT
- The NF-κB Factor RelB and Histone H3 Lysine Methyltransferase G9a Directly Interact to Generate Epigenetic Silencing in Endotoxin Tolerance
- (2009) Xiaoping Chen et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- RNA-binding Motif Protein 4 Translocates to Cytoplasmic Granules and Suppresses Translation via Argonaute2 during Muscle Cell Differentiation
- (2009) Jung-Chun Lin et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Dynamic and Selective Nucleosome Repositioning during Endotoxin Tolerance
- (2009) Mohamed El Gazzar et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- miR-146a Is Critical for Endotoxin-induced Tolerance
- (2009) Md A. Nahid et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Potent Phagocytic Activity with Impaired Antigen Presentation Identifying Lipopolysaccharide-Tolerant Human Monocytes: Demonstration in Isolated Monocytes from Cystic Fibrosis Patients
- (2009) Carlos del Fresno et al. JOURNAL OF IMMUNOLOGY
- Editorial: A recipe for inflammation
- (2009) Marco E. Bianchi JOURNAL OF LEUKOCYTE BIOLOGY
- Chromatin-Specific Remodeling by HMGB1 and Linker Histone H1 Silences Proinflammatory Genes during Endotoxin Tolerance
- (2009) M. E. Gazzar et al. MOLECULAR AND CELLULAR BIOLOGY
- The sepsis seesaw: tilting toward immunosuppression
- (2009) Richard S Hotchkiss et al. NATURE MEDICINE
- Endotoxin tolerance: new mechanisms, molecules and clinical significance
- (2009) Subhra K. Biswas et al. TRENDS IN IMMUNOLOGY
- Phosphorylation of Argonaute 2 at serine-387 facilitates its localization to processing bodies
- (2008) Yan Zeng et al. BIOCHEMICAL JOURNAL
- G9a and HP1 Couple Histone and DNA Methylation to TNFα Transcription Silencing during Endotoxin Tolerance
- (2008) Mohamed El Gazzar et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight?
- (2008) Witold Filipowicz et al. NATURE REVIEWS GENETICS
- A genome-wide analysis of LPS tolerance in macrophages
- (2007) Jörg Mages et al. IMMUNOBIOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More