The NF-κB Factor RelB and Histone H3 Lysine Methyltransferase G9a Directly Interact to Generate Epigenetic Silencing in Endotoxin Tolerance

Dynamic Histone H1 Isotype 4 Methylation and Demethylation by Histone Lysine Methyltransferase G9a/KMT1C and the Jumonji Domain-containing JMJD2/KDM4 Proteins

(2009) Patrick Trojer et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Chromatin-Specific Remodeling by HMGB1 and Linker Histone H1 Silences Proinflammatory Genes during Endotoxin Tolerance

(2009) M. E. Gazzar et al.   MOLECULAR AND CELLULAR BIOLOGY

Epigenetic Regulation in Human Brain—Focus on Histone Lysine Methylation

(2008) Schahram Akbarian et al.   BIOLOGICAL PSYCHIATRY

Epigenetic mechanisms in mammals

(2008) J. K. Kim et al.   CELLULAR AND MOLECULAR LIFE SCIENCES

RelB Sustains I B  Expression during Endotoxin Tolerance

(2008) X. Chen et al.   Clinical and Vaccine Immunology

Stabilization of RelB Requires Multidomain Interactions with p100/p52

(2008) Amanda J. Fusco et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

G9a and HP1 Couple Histone and DNA Methylation to TNFα Transcription Silencing during Endotoxin Tolerance

(2008) Mohamed El Gazzar et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Chromatin remodeling, development and disease

(2008) Myunggon Ko et al.   MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS

Methylation and deamination of CpGs generate p53-binding sites on a genomic scale

(2008) Tomasz Zemojtel et al.   TRENDS IN GENETICS