Amyotrophic lateral sclerosis-related VAPB P56S mutation differentially affects the function and survival of corticospinal and spinal motor neurons
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Title
Amyotrophic lateral sclerosis-related VAPB P56S mutation differentially affects the function and survival of corticospinal and spinal motor neurons
Authors
Keywords
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Journal
HUMAN MOLECULAR GENETICS
Volume 22, Issue 21, Pages 4293-4305
Publisher
Oxford University Press (OUP)
Online
2013-06-15
DOI
10.1093/hmg/ddt279
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Note: Only part of the references are listed.- Widespread aggregation of mutant VAPB associated with ALS does not cause motor neuron degeneration or modulate mutant SOD1 aggregation and toxicity in mice
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- Leucine-Rich Repeat Kinase 2 Regulates the Progression of Neuropathology Induced by Parkinson's-Disease-Related Mutant α-synuclein
- (2009) Xian Lin et al. NEURON
- A Cluster of Cholinergic Premotor Interneurons Modulates Mouse Locomotor Activity
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- From ER to Eph Receptors: New Roles for VAP Fragments
- (2008) Susan L. Ackerman et al. CELL
- The Amyotrophic Lateral Sclerosis 8 Protein VAPB Is Cleaved, Secreted, and Acts as a Ligand for Eph Receptors
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- VAPB interacts with and modulates the activity of ATF6
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- ALS-linked P56S-VAPB, an aggregated loss-of-function mutant of VAPB, predisposes motor neurons to ER stress-related death by inducing aggregation of co-expressed wild-type VAPB
- (2008) Hiroaki Suzuki et al. JOURNAL OF NEUROCHEMISTRY
- Progressive behavioral deficits in DJ-1-deficient mice are associated with normal nigrostriatal function
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