Pathological functions of interleukin-22 in chronic liver inflammation and fibrosis with hepatitis B virus infection by promoting T helper 17 cell recruitment
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Title
Pathological functions of interleukin-22 in chronic liver inflammation and fibrosis with hepatitis B virus infection by promoting T helper 17 cell recruitment
Authors
Keywords
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Journal
HEPATOLOGY
Volume 59, Issue 4, Pages 1331-1342
Publisher
Wiley
Online
2013-10-29
DOI
10.1002/hep.26916
References
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Related references
Note: Only part of the references are listed.- IL-22, not simply a Th17 cytokine
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- Recent advances in IL-22 biology
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- Interleukin-22: A cytokine produced by T, NK and NKT cell subsets, with importance in the innate immune defense and tissue protection
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- Interleukin-22 treatment ameliorates alcoholic liver injury in a murine model of chronic-binge ethanol feeding: Role of signal transducer and activator of transcription 3
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- Stage 3 immature human natural killer cells found in secondary lymphoid tissue constitutively and selectively express the TH17 cytokine interleukin-22
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- Interleukin-17-producing CD4+ T cells increase with severity of liver damage in patients with chronic hepatitis B
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- The Biological Functions of T Helper 17 Cell Effector Cytokines in Inflammation
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