标题
The voltage-gated sodium channel NaV1.9 in visceral pain
作者
关键词
-
出版物
NEUROGASTROENTEROLOGY AND MOTILITY
Volume 28, Issue 3, Pages 316-326
出版商
Wiley
发表日期
2015-10-14
DOI
10.1111/nmo.12698
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Discovery and Optimization of Selective Nav1.8 Modulator Series That Demonstrate Efficacy in Preclinical Models of Pain
- (2015) Sharan K. Bagal et al. ACS Medicinal Chemistry Letters
- Voltage-gated sodium channel mutations and painful neuropathy: Nav1.9 joins the family
- (2014) David L. H. Bennett BRAIN
- Gain-of-function mutations in sodium channel NaV1.9 in painful neuropathy
- (2014) Jianying Huang et al. BRAIN
- A Monoclonal Antibody that Targets a NaV1.7 Channel Voltage Sensor for Pain and Itch Relief
- (2014) Jun-Ho Lee et al. CELL
- Loss-of-Function of the Voltage-Gated Sodium Channel NaV1.5 (Channelopathies) in Patients With Irritable Bowel Syndrome
- (2014) Arthur Beyder et al. GASTROENTEROLOGY
- Specialized Functions of Nav1.5 and Nav1.9 Channels in Electrogenesis of Myenteric Neurons in Intact Mouse Ganglia
- (2014) N. Osorio et al. JOURNAL OF NEUROSCIENCE
- Multiple roles for NaV1.9 in the activation of visceral afferents by noxious inflammatory, mechanical, and human disease–derived stimuli
- (2014) James R.F. Hockley et al. PAIN
- Gain-of-Function Mutations in SCN11A Cause Familial Episodic Pain
- (2013) Xiang Yang Zhang et al. AMERICAN JOURNAL OF HUMAN GENETICS
- Mechanism of sodium channel NaV1.9 potentiation by G-protein signaling
- (2013) Carlos G. Vanoye et al. JOURNAL OF GENERAL PHYSIOLOGY
- A de novo gain-of-function mutation in SCN11A causes loss of pain perception
- (2013) Enrico Leipold et al. NATURE GENETICS
- Extrinsic primary afferent signalling in the gut
- (2013) Simon J. H. Brookes et al. Nature Reviews Gastroenterology & Hepatology
- The Nav1.9 channel regulates colonic motility in mice
- (2013) Carine Copel et al. Frontiers in Neuroscience
- Altered colorectal afferent function associated with TNBS-induced visceral hypersensitivity in mice
- (2012) Bin Feng et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- Long-term sensitization of mechanosensitive and -insensitive afferents in mice with persistent colorectal hypersensitivity
- (2012) Bin Feng et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- Translational neuropharmacology: the use of human isolated gastrointestinal tissues
- (2012) GJ Sanger et al. BRITISH JOURNAL OF PHARMACOLOGY
- Role of Nav1.9 in activity-dependent axon growth in motoneurons
- (2012) Narayan Subramanian et al. HUMAN MOLECULAR GENETICS
- Gain-of-function Nav1.8 mutations in painful neuropathy
- (2012) C. G. Faber et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Achieving translation in models of visceral pain
- (2011) David Colin Bulmer et al. CURRENT OPINION IN PHARMACOLOGY
- Guidelines for the management of inflammatory bowel disease in adults
- (2011) C. Mowat et al. GUT
- Nav1.9 Channel Contributes to Mechanical and Heat Pain Hypersensitivity Induced by Subacute and Chronic Inflammation
- (2011) Stéphane Lolignier et al. PLoS One
- Characterization of silent afferents in the pelvic and splanchnic innervations of the mouse colorectum
- (2010) Bin Feng et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- Sodium Channels in Normal and Pathological Pain
- (2010) Sulayman D. Dib-Hajj et al. Annual Review of Neuroscience
- Familial pain syndromes from mutations of the Nav1.7 sodium channel
- (2009) Tanya Z. Fischer et al. Annals of the New York Academy of Sciences
- Exploring the role of nociceptor-specific sodium channels in pain transmission using Nav1.8 and Nav1.9 knockout mice
- (2009) Sandra Leo et al. BEHAVIOURAL BRAIN RESEARCH
- Post-inflammatory colonic afferent sensitisation: different subtypes, different pathways and different time courses
- (2009) P A Hughes et al. GUT
- Pain and inflammatory bowel disease
- (2009) Klaus Bielefeldt et al. INFLAMMATORY BOWEL DISEASES
- Trinitrobenzenesulphonic acid colitis alters Nav1.8 channel expression in mouse dorsal root ganglia neurons
- (2009) d. e. king et al. NEUROGASTROENTEROLOGY AND MOTILITY
- The voltage-gated sodium channel Nav1.9 is required for inflammation-based urinary bladder dysfunction
- (2009) Amy M. Ritter et al. NEUROSCIENCE LETTERS
- Basic and clinical aspects of gastrointestinal pain
- (2009) Charles H. Knowles et al. PAIN
- The Functional–Organic Dichotomy: Postinfectious Irritable Bowel Syndrome and Inflammatory Bowel Disease–Irritable Bowel Syndrome
- (2008) Madhusudan Grover et al. Clinical Gastroenterology and Hepatology
- Lack of colonic inflammation-induced acute visceral hypersensitivity to colorectal distension in Nav1.9 knockout mice
- (2008) V. Martinez et al. EUROPEAN JOURNAL OF PAIN
- Comparative study of the distribution of the α-subunits of voltage-gated sodium channels in normal and axotomized rat dorsal root ganglion neurons
- (2008) Tetsuo Fukuoka et al. JOURNAL OF COMPARATIVE NEUROLOGY
- Inflammatory Mediators Increase Nav1.9 Current and Excitability in Nociceptors through a Coincident Detection Mechanism
- (2008) François Maingret et al. JOURNAL OF GENERAL PHYSIOLOGY
- GTP up-regulated persistent Na+current and enhanced nociceptor excitability require NaV1.9
- (2007) Johan A. R. Östman et al. JOURNAL OF PHYSIOLOGY-LONDON
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