Nitric oxide (•NO) generation but not ROS plays a major role in silibinin-induced autophagic and apoptotic death in human epidermoid carcinoma A431 cells
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Nitric oxide (•NO) generation but not ROS plays a major role in silibinin-induced autophagic and apoptotic death in human epidermoid carcinoma A431 cells
Authors
Keywords
-
Journal
FREE RADICAL RESEARCH
Volume 46, Issue 11, Pages 1346-1360
Publisher
Informa UK Limited
Online
2012-08-06
DOI
10.3109/10715762.2012.715369
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Autophagy following heat stress: The role of aging and protein nitration
- (2014) Jamie M. Swanlund et al. Autophagy
- Is mitochondrial generation of reactive oxygen species a trigger for autophagy?
- (2014) Yongqiang Chen et al. Autophagy
- P53 activation plays a crucial role in silibinin induced ROS generation via PUMA and JNK
- (2012) Simiao Fan et al. FREE RADICAL RESEARCH
- Silibinin triggers apoptotic signaling pathways and autophagic survival response in human colon adenocarcinoma cells and their derived metastatic cells
- (2011) Henriette Kauntz et al. APOPTOSIS
- Role of ROS in the protective effect of silibinin on sodium nitroprusside-induced apoptosis in rat pheochromocytoma PC12 cells
- (2011) Binbin Liu et al. FREE RADICAL RESEARCH
- Silibinin induced-autophagic and apoptotic death is associated with an increase in reactive oxygen and nitrogen species in HeLa cells
- (2011) Simiao Fan et al. FREE RADICAL RESEARCH
- Antioxidant, DNA cleavage, and cellular effects of silibinin and a new oxovanadium(IV)/silibinin complex
- (2011) Luciana G. Naso et al. JOURNAL OF BIOLOGICAL INORGANIC CHEMISTRY
- Role of oxidative/nitrosative stress-mediated Bcl-2 regulation in apoptosis and malignant transformation
- (2010) Neelam Azad et al. Annals of the New York Academy of Sciences
- Mutant p53 exhibits trivial effects on mitochondrial functions which can be reactivated by ellipticine in lymphoma cells
- (2010) Fei Wang et al. APOPTOSIS
- Silibinin Induced Autophagic and Apoptotic Cell Death in HT1080 Cells Through a Reactive Oxygen Species Pathway
- (2010) Wenjun Duan et al. JOURNAL OF PHARMACOLOGICAL SCIENCES
- Silymarin protects spinal cord and cortical cells against oxidative stress and lipopolysaccharide stimulation
- (2010) May-Jywan Tsai et al. NEUROCHEMISTRY INTERNATIONAL
- Inhibiting NF-κB activation and ROS production are involved in the mechanism of silibinin's protection against D-galactose-induced senescence
- (2010) Qiong Wang et al. PHARMACOLOGY BIOCHEMISTRY AND BEHAVIOR
- Silibinin attenuates cognitive deficits and decreases of dopamine and serotonin induced by repeated methamphetamine treatment
- (2009) Ping Lu et al. BEHAVIOURAL BRAIN RESEARCH
- Silibinin prevents amyloid β peptide-induced memory impairment and oxidative stress in mice
- (2009) P Lu et al. BRITISH JOURNAL OF PHARMACOLOGY
- Bid integrates intrinsic and extrinsic signaling in apoptosis induced by α-tocopheryl succinate in human gastric carcinoma cells
- (2009) Yan Zhao et al. CANCER LETTERS
- Silibinin induces protective superoxide generation in human breast cancer MCF-7 cells
- (2009) Hong-Jun Wang et al. FREE RADICAL RESEARCH
- The Protective Effect of Silibinin Against Mitomycin C–Induced Intrinsic Apoptosis in Human Melanoma A375-S2 Cells
- (2009) Yuan-yuan Jiang et al. JOURNAL OF PHARMACOLOGICAL SCIENCES
- Silibinin Attenuates Amyloid 25-35 Peptide-Induced Memory Impairments: Implication of Inducible Nitric-Oxide Synthase and Tumor Necrosis Factor- in Mice
- (2009) P. Lu et al. JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS
- Evaluation of silibinin on the viability, migration and adhesion of the human prostate adenocarcinoma (PC-3) cell line
- (2008) M MOKHTARI et al. CELL BIOLOGY INTERNATIONAL
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreDiscover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversation