Acclimatization of skeletal muscle mitochondria to high-altitude hypoxia during an ascent of Everest

Hypoxia. 2. Hypoxia regulates cellular metabolism

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Survival in Critical Illness Is Associated with Early Activation of Mitochondrial Biogenesis

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Cardiac response to hypobaric hypoxia: persistent changes in cardiac mass, function, and energy metabolism after a trek to Mt. Everest Base Camp

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PKCε promotes cardiac mitochondrial and metabolic adaptation to chronic hypobaric hypoxia by GSK3β inhibition

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Mechanisms of mitophagy

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Hypoxia-regulated microRNA-210 modulates mitochondrial function and decreases ISCU and COX10 expression

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MicroRNA-210 Regulates Mitochondrial Free Radical Response to Hypoxia and Krebs Cycle in Cancer Cells by Targeting Iron Sulfur Cluster Protein ISCU

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The Effect of High-Altitude on Human Skeletal Muscle Energetics: 31P-MRS Results from the Caudwell Xtreme Everest Expedition

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MicroRNA-210 Controls Mitochondrial Metabolism during Hypoxia by Repressing the Iron-Sulfur Cluster Assembly Proteins ISCU1/2

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Deterioration of physical performance and cognitive function in rats with short-term high-fat feeding

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Arterial Blood Gases and Oxygen Content in Climbers on Mount Everest

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Metabolic adaptation of skeletal muscle to high altitude hypoxia: how new technologies could resolve the controversies

(2009) Andrew J Murray   Genome Medicine

Mountainside to bedside: reality or fiction?

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ATF-1 Is a Hypoxia-responsive Transcriptional Activator of Skeletal Muscle Mitochondrial-uncoupling Protein 3

(2008) Zhongping Lu et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Increased mitochondrial uncoupling proteins, respiratory uncoupling and decreased efficiency in the chronically infarcted rat heart

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Deficiency or inhibition of oxygen sensor Phd1 induces hypoxia tolerance by reprogramming basal metabolism

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