The response of neuromuscular junctions to injury is developmentally regulated

Using mouse cranial muscles to investigate neuromuscular pathology in vivo

(2010) L.M. Murray et al.   NEUROMUSCULAR DISORDERS

Review: Neuromuscular synaptic vulnerability in motor neurone disease: amyotrophic lateral sclerosis and spinal muscular atrophy

(2010) L. M. Murray et al.   NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY

Pre-symptomatic development of lower motor neuron connectivity in a mouse model of severe spinal muscular atrophy

(2009) Lyndsay M. Murray et al.   HUMAN MOLECULAR GENETICS

Impaired Synaptic Vesicle Release and Immaturity of Neuromuscular Junctions in Spinal Muscular Atrophy Mice

(2009) L. Kong et al.   JOURNAL OF NEUROSCIENCE

Alternative Splicing Events Are a Late Feature of Pathology in a Mouse Model of Spinal Muscular Atrophy

(2009) Dirk Bäumer et al.   PLoS Genetics

Rapid loss of motor nerve terminals following hypoxia–reperfusion injury occurs via mechanisms distinct from classic Wallerian degeneration

(2008) Becki Baxter et al.   JOURNAL OF ANATOMY

Loss of translation elongation factor (eEF1A2) expressionin vivodifferentiates between Wallerian degeneration and dying-back neuronal pathology

(2008) Lyndsay M. Murray et al.   JOURNAL OF ANATOMY

The role of complement and complement regulators in mediating motor nerve terminal injury in murine models of Guillain–Barré syndrome

(2008) Hugh J. Willison et al.   JOURNAL OF NEUROIMMUNOLOGY