Complex environment experience rescues impaired neurogenesis, enhances synaptic plasticity, and attenuates neuropathology in familial Alzheimer’s disease-linked APPswe/PS1ΔE9 mice
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Title
Complex environment experience rescues impaired neurogenesis, enhances synaptic plasticity, and attenuates neuropathology in familial Alzheimer’s disease-linked APPswe/PS1ΔE9 mice
Authors
Keywords
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Journal
FASEB JOURNAL
Volume 24, Issue 6, Pages 1667-1681
Publisher
FASEB
Online
2010-01-20
DOI
10.1096/fj.09-136945
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Note: Only part of the references are listed.- Impaired neurogenesis is an early event in the etiology of familial Alzheimer's disease in transgenic mice
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- The amino terminus of tau inhibits kinesin-dependent axonal transport: Implications for filament toxicity
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- Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory
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- Non-Cell-Autonomous Effects of Presenilin 1 Variants on Enrichment-Mediated Hippocampal Progenitor Cell Proliferation and Differentiation
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- Differential regulation of neurogenesis in two neurogenic regions of APPswe/PS1dE9 transgenic mice
- (2008) Tetsuhiro Niidome et al. NEUROREPORT
- Impaired Adult Neurogenesis in the Dentate Gyrus of a Triple Transgenic Mouse Model of Alzheimer's Disease
- (2008) José J. Rodríguez et al. PLoS One
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