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Title
Innate antimicrobial immunity in inflammatory bowel diseases
Authors
Keywords
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Journal
Expert Review of Clinical Immunology
Volume 6, Issue 5, Pages 809-818
Publisher
Informa UK Limited
Online
2010-09-09
DOI
10.1586/eci.10.56
References
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- (2009) Jesus K. Yamamoto-Furusho et al. INFLAMMATORY BOWEL DISEASES
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