Targeting amyloid-beta by glucagon-like peptide -1 (GLP-1) in Alzheimer's disease and diabetes
Published 2011 View Full Article
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Title
Targeting amyloid-beta by glucagon-like peptide -1 (GLP-1) in Alzheimer's disease and diabetes
Authors
Keywords
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Journal
EXPERT OPINION ON THERAPEUTIC TARGETS
Volume 15, Issue 10, Pages 1153-1162
Publisher
Informa Healthcare
Online
2011-07-13
DOI
10.1517/14728222.2011.600691
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- Long-term inhibition of dipeptidyl peptidase-4 in Alzheimer’s prone mice
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- GLP-1 receptor stimulation preserves primary cortical and dopaminergic neurons in cellular and rodent models of stroke and Parkinsonism
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- GLP-1 agonists facilitate hippocampal LTP and reverse the impairment of LTP induced by beta-amyloid
- (2008) Victor A. Gault et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Glucagon-like peptide 1 receptor stimulation by exendin-4 reverses key deficits in distinct rodent models of Parkinson's disease
- (2008) Alexander Harkavyi et al. Journal of Neuroinflammation
- Beta amyloid and hyperphosphorylated tau deposits in the pancreas in type 2 diabetes
- (2008) Judith Miklossy et al. NEUROBIOLOGY OF AGING
- Mutated recombinant human glucagon-like peptide-1 protects SH-SY5Y cells from apoptosis induced by amyloid-β peptide (1–42)
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- Peptide hormone exendin-4 stimulates subventricular zone neurogenesis in the adult rodent brain and induces recovery in an animal model of parkinson's disease
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