Increased frequencies of IL-31-producing T cells are found in chronic atopic dermatitis skin

Low Expression of the IL-23/Th17 Pathway in Atopic Dermatitis Compared to Psoriasis

(2014) E. Guttman-Yassky et al.   JOURNAL OF IMMUNOLOGY

IL-31 Expression by Inflammatory Cells is Preferentially Elevated in Atopic Dermatitis

(2012) GFL Hofbauer et al.   ACTA DERMATO-VENEREOLOGICA

IL-31 regulates differentiation and filaggrin expression in human organotypic skin models

(2011) Christian Cornelissen et al.   JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY

Mutual Antagonism of T Cells Causing Psoriasis and Atopic Eczema

(2011) Stefanie Eyerich et al.   NEW ENGLAND JOURNAL OF MEDICINE

Cowhage-Induced Itch as an Experimental Model for Pruritus. A Comparative Study with Histamine-Induced Itch

(2011) Alexandru D. P. Papoiu et al.   PLoS One

IL-17 and IL-22 in atopic allergic disease

(2010) Yuri Souwer et al.   CURRENT OPINION IN IMMUNOLOGY

Increased levels of serum IL-31 in chronic spontaneous urticaria

(2010) Ulrike Raap et al.   EXPERIMENTAL DERMATOLOGY

Mechanical injury polarizes skin dendritic cells to elicit a TH2 response by inducing cutaneous thymic stromal lymphopoietin expression

(2010) Michiko K. Oyoshi et al.   JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY

Decrease in circulating Th17 cells correlates with increased levels of CCL17, IgE and eosinophils in atopic dermatitis

(2010) Sayaka Hayashida et al.   JOURNAL OF DERMATOLOGICAL SCIENCE

Cathepsin S Elicits Itch and Signals via Protease-Activated Receptors

(2010) Vemuri B. Reddy et al.   JOURNAL OF INVESTIGATIVE DERMATOLOGY

Serum measurement of interleukin-31 (IL-31) in paediatric atopic dermatitis: elevated levels correlate with severity scoring

(2010) MHM Ezzat et al.   JOURNAL OF THE EUROPEAN ACADEMY OF DERMATOLOGY AND VENEREOLOGY

Overrepresentation of IL-17A and IL-22 Producing CD8 T Cells in Lesional Skin Suggests Their Involvement in the Pathogenesis of Psoriasis

(2010) Pieter C. M. Res et al.   PLoS One

New insight into mechanisms of pruritus from molecular studies on familial primary localized cutaneous amyloidosis

(2009) A. Tanaka et al.   BRITISH JOURNAL OF DERMATOLOGY

Precarious Balance: Th17 Cells in Host Defense

(2009) A. Peck et al.   INFECTION AND IMMUNITY

Cytokine modulation of atopic dermatitis filaggrin skin expression

(2009) Michael D. Howell et al.   JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY

IL-22–producing “T22” T cells account for upregulated IL-22 in atopic dermatitis despite reduced IL-17–producing TH17 T cells

(2009) Kristine E. Nograles et al.   JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY

Identification of a human helper T cell population that has abundant production of interleukin 22 and is distinct from TH-17, TH1 and TH2 cells

(2009) Sara Trifari et al.   NATURE IMMUNOLOGY

Production of interleukin 22 but not interleukin 17 by a subset of human skin-homing memory T cells

(2009) Thomas Duhen et al.   NATURE IMMUNOLOGY

Loss-of-function polymorphisms in the filaggrin gene are associated with an increased susceptibility to chronic irritant contact dermatitis: a case-control study

(2008) C.M. de Jongh et al.   BRITISH JOURNAL OF DERMATOLOGY

Structures and biological functions of IL-31 and IL-31 receptors

(2008) Qing Zhang et al.   CYTOKINE & GROWTH FACTOR REVIEWS

Anti-interleukin-31-antibodies ameliorate scratching behaviour in NC/Nga mice: a model of atopic dermatitis

(2008) Øystein Grimstad et al.   EXPERIMENTAL DERMATOLOGY

Filaggrin null mutations are associated with atopic dermatitis and elevated levels of IgE in the Japanese population: a family and case–control study

(2008) Hisako Enomoto et al.   JOURNAL OF HUMAN GENETICS

Possible Pathogenic Role of Th17 Cells for Atopic Dermatitis

(2008) Chizuko Koga et al.   JOURNAL OF INVESTIGATIVE DERMATOLOGY

Loss-of-Function Mutations in the Filaggrin Gene Lead to Reduced Level of Natural Moisturizing Factor in the Stratum Corneum

(2008) Sanja Kezic et al.   JOURNAL OF INVESTIGATIVE DERMATOLOGY

Atopic Dermatitis

(2008) Thomas Bieber   NEW ENGLAND JOURNAL OF MEDICINE