Astragaloside IV inhibits renal tubulointerstitial fibrosis by blocking TGF-β/Smad signaling pathway in vivo and in vitro
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Astragaloside IV inhibits renal tubulointerstitial fibrosis by blocking TGF-β/Smad signaling pathway in vivo and in vitro
Authors
Keywords
-
Journal
EXPERIMENTAL BIOLOGY AND MEDICINE
Volume 239, Issue 10, Pages 1310-1324
Publisher
SAGE Publications
Online
2014-05-31
DOI
10.1177/1535370214532597
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Astragaloside IV prevents acute kidney injury in two rodent models by inhibiting oxidative stress and apoptosis pathways
- (2013) Dingkun Gui et al. APOPTOSIS
- Astragaloside IV ameliorates renal injury in streptozotocin-induced diabetic rats through inhibiting NF-κB-mediated inflammatory genes expression
- (2013) Dingkun Gui et al. CYTOKINE
- Curcumin Inhibits Transforming Growth Factor-β1-Induced EMT via PPARγ Pathway, Not Smad Pathway in Renal Tubular Epithelial Cells
- (2013) Rui Li et al. PLoS One
- Disruption of Smad7 Promotes ANG II-Mediated Renal Inflammation and Fibrosis via Sp1-TGF-β/Smad3-NF.κB-Dependent Mechanisms in Mice
- (2013) Guan-Xian Liu et al. PLoS One
- Norcantharidin Inhibits Renal Interstitial Fibrosis by Blocking the Tubular Epithelial-Mesenchymal Transition
- (2013) Ying Li et al. PLoS One
- Asiatic Acid Inhibits Liver Fibrosis by Blocking TGF-beta/Smad Signaling In Vivo and In Vitro
- (2012) Li-xia Tang et al. PLoS One
- Astragaloside IV, a Novel Antioxidant, Prevents Glucose-Induced Podocyte Apoptosis In Vitro and In Vivo
- (2012) Dingkun Gui et al. PLoS One
- Therapeutic Effects of Astragaloside IV on Myocardial Injuries: Multi-Target Identification and Network Analysis
- (2012) Jing Zhao et al. PLoS One
- Dioscorea alata Attenuates Renal Interstitial Cellular Fibrosis by Regulating Smad- and Epithelial-Mesenchymal Transition Signaling Pathways
- (2012) Shu-Fen Liu et al. PLoS One
- Astragaloside IV synergizes with ferulic acid to inhibit renal tubulointerstitial fibrosis in rats with obstructive nephropathy
- (2011) LQ Meng et al. BRITISH JOURNAL OF PHARMACOLOGY
- Epithelial-mesenchymal transition in renal fibrosis - evidence for and against
- (2011) Maria Fragiadaki et al. INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY
- Suramin Inhibits Renal Fibrosis in Chronic Kidney Disease
- (2011) N. Liu et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Fluorofenidone attenuates renal interstitial fibrosis in the rat model of obstructive nephropathy
- (2011) Bing-xin Li et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Astragaloside IV Attenuates Complement Membranous Attack Complex Induced Podocyte Injury Through the MAPK Pathway
- (2011) Rong Zheng et al. PHYTOTHERAPY RESEARCH
- Astragaloside IV Improves Homocysteine-Induced Acute Phase Endothelial Dysfunction via Antioxidation
- (2010) Li-Hong Qiu et al. BIOLOGICAL & PHARMACEUTICAL BULLETIN
- Mechanisms of Tubulointerstitial Fibrosis
- (2010) M. Zeisberg et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Smad2 Protects against TGF- /Smad3-Mediated Renal Fibrosis
- (2010) X. M. Meng et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Advanced Glycation End-Products Induce Tubular CTGF via TGF- -Independent Smad3 Signaling
- (2009) A. C.K. Chung et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Transglutaminase inhibition ameliorates experimental diabetic nephropathy
- (2009) Linghong Huang et al. KIDNEY INTERNATIONAL
- Smad7 as a therapeutic agent for chronic kidney diseases
- (2008) Hui Yao Lan Frontiers in Bioscience-Landmark
- Disruption of the Smad7 gene promotes renal fibrosis and inflammation in unilateral ureteral obstruction (UUO) in mice
- (2008) A. C. K. Chung et al. NEPHROLOGY DIALYSIS TRANSPLANTATION
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started