Converse regulation of CCR7-driven human dendritic cell migration by prostaglandin E2and liver X receptor activation
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Converse regulation of CCR7-driven human dendritic cell migration by prostaglandin E2and liver X receptor activation
Authors
Keywords
-
Journal
EUROPEAN JOURNAL OF IMMUNOLOGY
Volume 42, Issue 11, Pages 2949-2958
Publisher
Wiley
Online
2012-08-14
DOI
10.1002/eji.201242523
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- In Vitro Differentiation of Dendritic Cells in the Presence of Prostaglandin E2 Alters the IL-12/IL-23 Balance and Promotes Differentiation of Th17 Cells
- (2014) T. Khayrullina et al. JOURNAL OF IMMUNOLOGY
- Positive feedback between PGE2 and COX2 redirects the differentiation of human dendritic cells toward stable myeloid-derived suppressor cells
- (2011) N. Obermajer et al. BLOOD
- Fascin1 Promotes Cell Migration of Mature Dendritic Cells
- (2011) Y. Yamakita et al. JOURNAL OF IMMUNOLOGY
- PGE2 transiently enhances DC expression of CCR7 but inhibits the ability of DCs to produce CCL19 and attract naive T cells
- (2010) R. Muthuswamy et al. BLOOD
- LXR promotes the maximal egress of monocyte-derived cells from mouse aortic plaques during atherosclerosis regression
- (2010) Jonathan E. Feig et al. JOURNAL OF CLINICAL INVESTIGATION
- Activation of Liver X Receptor Sensitizes Human Dendritic Cells to Inflammatory Stimuli
- (2010) D. Torocsik et al. JOURNAL OF IMMUNOLOGY
- Genetic deletion of mPGES-1 abolishes PGE2 production in murine dendritic cells and alters the cytokine profile, but does not affect maturation or migration
- (2010) S.U. Monrad et al. PROSTAGLANDINS LEUKOTRIENES AND ESSENTIAL FATTY ACIDS
- PPARγ and LXR Signaling Inhibit Dendritic Cell-Mediated HIV-1 Capture and trans-Infection
- (2010) Timothy M. Hanley et al. PLoS Pathogens
- MSCs inhibit monocyte-derived DC maturation and function by selectively interfering with the generation of immature DCs: central role of MSC-derived prostaglandin E2
- (2009) G. M. Spaggiari et al. BLOOD
- Prostaglandin E2 at new glance: Novel insights in functional diversity offer therapeutic chances
- (2009) Daniel F. Legler et al. INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY
- Prostaglandin E2 regulates Th17 cell differentiation and function through cyclic AMP and EP2/EP4 receptor signaling
- (2009) Katia Boniface et al. JOURNAL OF EXPERIMENTAL MEDICINE
- CCR7-specific migration to CCL19 and CCL21 is induced by PGE2 stimulation in human monocytes: Involvement of EP2/EP4 receptors activation
- (2009) Sandra C. Côté et al. MOLECULAR IMMUNOLOGY
- Tumor-mediated liver X receptor-α activation inhibits CC chemokine receptor-7 expression on dendritic cells and dampens antitumor responses
- (2009) Eduardo J Villablanca et al. NATURE MEDICINE
- Prostaglandin E2–EP4 signaling promotes immune inflammation through TH1 cell differentiation and TH17 cell expansion
- (2009) Chengcan Yao et al. NATURE MEDICINE
- Targeted lipidomics reveals mPGES-1-PGE2 as a therapeutic target for multiple sclerosis
- (2009) Y. Kihara et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Prostaglandin E2 enhances T-cell proliferation by inducing the costimulatory molecules OX40L, CD70, and 4-1BBL on dendritic cells
- (2008) P. Krause et al. BLOOD
- Mechanisms and Consequences of Dendritic Cell Migration
- (2008) David Alvarez et al. IMMUNITY
- Rapid leukocyte migration by integrin-independent flowing and squeezing
- (2008) Tim Lämmermann et al. NATURE
- Integration of metabolism and inflammation by lipid-activated nuclear receptors
- (2008) Steven J. Bensinger et al. NATURE
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started