The tryptophan metabolite 3-hydroxyanthranilic acid lowers plasma lipids and decreases atherosclerosis in hypercholesterolaemic mice
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
The tryptophan metabolite 3-hydroxyanthranilic acid lowers plasma lipids and decreases atherosclerosis in hypercholesterolaemic mice
Authors
Keywords
-
Journal
EUROPEAN HEART JOURNAL
Volume 33, Issue 16, Pages 2025-2034
Publisher
Oxford University Press (OUP)
Online
2012-06-19
DOI
10.1093/eurheartj/ehs175
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Role of macrophage scavenger receptors in atherosclerosis
- (2012) Julia Kzhyshkowska et al. IMMUNOBIOLOGY
- The Tryptophan Metabolite 3-Hydroxyanthranilic Acid Plays Anti-Inflammatory and Neuroprotective Roles During Inflammation
- (2011) Daniela Krause et al. AMERICAN JOURNAL OF PATHOLOGY
- Identification of a Danger-Associated Peptide From Apolipoprotein B100 (ApoBDS-1) That Triggers Innate Proatherogenic Responses
- (2011) Daniel F.J. Ketelhuth et al. CIRCULATION
- The PPAR-RXR Transcriptional Complex in the Vasculature
- (2011) Jorge Plutzky CIRCULATION RESEARCH
- Plasmacytoid Dendritic Cells Protect Against Atherosclerosis by Tuning T-Cell Proliferation and Activity
- (2011) Isabelle T.M.N. Daissormont et al. CIRCULATION RESEARCH
- Triglyceride-rich lipoproteins and high-density lipoprotein cholesterol in patients at high risk of cardiovascular disease: evidence and guidance for management
- (2011) M. John Chapman et al. EUROPEAN HEART JOURNAL
- Nicotinic acid inhibits progression of atherosclerosis in mice through its receptor GPR109A expressed by immune cells
- (2011) Martina Lukasova et al. JOURNAL OF CLINICAL INVESTIGATION
- Cellular immunity, low-density lipoprotein and atherosclerosis: Break of tolerance in the artery wall
- (2011) Daniel Ketelhuth et al. THROMBOSIS AND HAEMOSTASIS
- Nuclear Receptors and Inflammation Control: Molecular Mechanisms and Pathophysiological Relevance
- (2010) Wendy Huang et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Control of immune response by amino acid metabolism
- (2010) Ursula Grohmann et al. IMMUNOLOGICAL REVIEWS
- Tryptophan metabolite 3-hydroxyanthranilic acid selectively induces activated T cell death via intracellular GSH depletion
- (2010) Sun-Mi Lee et al. IMMUNOLOGY LETTERS
- Dendritic Cells, Indoleamine 2,3 Dioxygenase and Acquired Immune Privilege
- (2010) Lei Huang et al. INTERNATIONAL REVIEWS OF IMMUNOLOGY
- Inhibition of T cell response to native low-density lipoprotein reduces atherosclerosis
- (2010) Andreas Hermansson et al. JOURNAL OF EXPERIMENTAL MEDICINE
- IDO Upregulates Regulatory T Cells via Tryptophan Catabolite and Suppresses Encephalitogenic T Cell Responses in Experimental Autoimmune Encephalomyelitis
- (2010) Y. Yan et al. JOURNAL OF IMMUNOLOGY
- Kynurenine is an endothelium-derived relaxing factor produced during inflammation
- (2010) Yutang Wang et al. NATURE MEDICINE
- Inactivation of the LRP1 Intracellular NPxYxxL Motif in LDLR-Deficient Mice Enhances Postprandial Dyslipidemia and Atherosclerosis
- (2009) Philip L.S.M. Gordts et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Suppression of T-cell response and prolongation of allograft survival in a rat model by tryptophan catabolites
- (2009) Xiangchen Dai et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- PPARδ ligand L-165041 ameliorates Western diet-induced hepatic lipid accumulation and inflammation in LDLR−/− mice
- (2009) Hyun-Joung Lim et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Role of PPAR-gamma in the Modulation of CD36 and FcgammaRII induced by LDL with Low and High Degrees of Oxidation During the Differentiation of the Monocytic THP-1 Cell Line
- (2008) Francisco J.O. Rios et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- Autoantibody Response to Chromatographic Fractions from Oxidized LDL in Unstable Angina Patients and Healthy Controls
- (2008) D. F. J. Ketelhuth et al. SCANDINAVIAN JOURNAL OF IMMUNOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started