Ras-related C3 botulinum toxin substrate 1 (RAC1) regulates glucose-stimulated insulin secretion via modulation of F-actin

Title
Ras-related C3 botulinum toxin substrate 1 (RAC1) regulates glucose-stimulated insulin secretion via modulation of F-actin
Authors
Keywords
Beta cell, Diabetes, F-actin, Insulin secretion, RAC1
Journal
DIABETOLOGIA
Volume 56, Issue 5, Pages 1088-1097
Publisher
Springer Nature
Online
2013-02-14
DOI
10.1007/s00125-013-2849-5

Ask authors/readers for more resources

Reprint

Contact the author

Find the ideal target journal for your manuscript

Explore over 38,000 international journals covering a vast array of academic fields.

Search

Ask a Question. Answer a Question.

Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.

Get Started