SMAD2 disruption in mouse pancreatic beta cells leads to islet hyperplasia and impaired insulin secretion due to the attenuation of ATP-sensitive K+ channel activity

Title
SMAD2 disruption in mouse pancreatic beta cells leads to islet hyperplasia and impaired insulin secretion due to the attenuation of ATP-sensitive K+ channel activity
Authors
Keywords
Insulin, K<sub>ATP</sub> channels, Pancreatic beta cells, SMAD2, TGF-β
Journal
DIABETOLOGIA
Volume 57, Issue 1, Pages 157-166
Publisher
Springer Nature
Online
2013-09-25
DOI
10.1007/s00125-013-3062-2

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