Activation of PI3K and R-ras signaling promotes the extension of sensory axons on inhibitory chondroitin sulfate proteoglycans

Functional regeneration beyond the glial scar

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Comparison of sensory neuron growth cone and filopodial responses to structurally diverse aggrecan variants, in vitro

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PTEN Inhibition to Facilitate Intrinsic Regenerative Outgrowth of Adult Peripheral Axons

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Intra-Axonal Translation of RhoA Promotes Axon Growth Inhibition by CSPG

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Nerve Growth Factor-Induced Formation of Axonal Filopodia and Collateral Branches Involves the Intra-Axonal Synthesis of Regulators of the Actin-Nucleating Arp2/3 Complex

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R-Ras controls axon branching through afadin in cortical neurons

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The actin nucleating Arp2/3 complex contributes to the formation of axonal filopodia and branches through the regulation of actin patch precursors to filopodia

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Leukocyte Common Antigen-Related Phosphatase Is a Functional Receptor for Chondroitin Sulfate Proteoglycan Axon Growth Inhibitors

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Proteoglycan-Specific Molecular Switch for RPTP  Clustering and Neuronal Extension

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Semaphorin 4D/Plexin-B1 Stimulates PTEN Activity through R-Ras GTPase-activating Protein Activity, Inducing Growth Cone Collapse in Hippocampal Neurons

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Nerve Growth Factor Induces Axonal Filopodia through Localized Microdomains of Phosphoinositide 3-Kinase Activity That Drive the Formation of Cytoskeletal Precursors to Filopodia

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PTEN deletion enhances the regenerative ability of adult corticospinal neurons

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Current Perspectives on Akt Akt-ivation and Akt-ions

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Semaphorin 3A inhibits ERM protein phosphorylation in growth cone filopodia through inactivation of PI3K

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The RSK factors of activating the Ras/MAPK signaling cascade

(2008) Audrey Carriere   Frontiers in Bioscience-Landmark

Choose your own path: specificity in Ras GTPase signaling

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Promoting Axon Regeneration in the Adult CNS by Modulation of the PTEN/mTOR Pathway

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