Neuronal Hyperexcitability: A Substrate for Central Neuropathic Pain After Spinal Cord Injury
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Title
Neuronal Hyperexcitability: A Substrate for Central Neuropathic Pain After Spinal Cord Injury
Authors
Keywords
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Journal
Current Pain and Headache Reports
Volume 15, Issue 3, Pages 215-222
Publisher
Springer Nature
Online
2011-03-09
DOI
10.1007/s11916-011-0186-2
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Note: Only part of the references are listed.- Mexiletine suppresses nodal persistent sodium currents in sensory axons of patients with neuropathic pain
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- Transplantation of GABAergic Neurons from ESCs Attenuates Tactile Hypersensitivity Following Spinal Cord Injury
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- Triggering genetically-expressed transneuronal tracers by peripheral axotomy reveals convergent and segregated sensory neuron-spinal cord connectivity
- (2009) J.M. Bráz et al. NEUROSCIENCE
- Peripheral and central sensitization in remote spinal cord regions contribute to central neuropathic pain after spinal cord injury
- (2009) Susan M. Carlton et al. PAIN
- Residual spinothalamic tract pathways predict development of central pain after spinal cord injury
- (2008) G. Wasner et al. BRAIN
- Activation of p38 MAP kinase is involved in central neuropathic pain following spinal cord injury
- (2008) Eric D. Crown et al. EXPERIMENTAL NEUROLOGY
- Remote activation of microglia and pro-inflammatory cytokines predict the onset and severity of below-level neuropathic pain after spinal cord injury in rats
- (2008) Megan Ryan Detloff et al. EXPERIMENTAL NEUROLOGY
- Neuropathic Pain Memory Is Maintained by Rac1-Regulated Dendritic Spine Remodeling after Spinal Cord Injury
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- Propentofylline attenuates allodynia, glial activation and modulates GABAergic tone after spinal cord injury in the rat
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- Nuclear factor-κB decoy amelioration of spinal cord injury-induced inflammation and behavior outcomes
- (2007) Danny Salah Rafati et al. JOURNAL OF NEUROSCIENCE RESEARCH
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