ROS generated during early reperfusion contribute to intermittent hypobaric hypoxia-afforded cardioprotection against postischemia-induced Ca2+ overload and contractile dysfunction via the JAK2/STAT3 pathway

Title
ROS generated during early reperfusion contribute to intermittent hypobaric hypoxia-afforded cardioprotection against postischemia-induced Ca2+ overload and contractile dysfunction via the JAK2/STAT3 pathway
Authors
Keywords
Reactive oxygen species, Intracellular Ca, concentration, STAT3, Bcl-2, SR Ca, -ATPase 2, Intermittent hypobaric hypoxia
Journal
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
Volume 81, Issue -, Pages 150-161
Publisher
Elsevier BV
Online
2015-02-28
DOI
10.1016/j.yjmcc.2015.02.015

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