Postprandial oxidative stress is modified by dietary fat: evidence from a human intervention study

Dietary fat differentially influences regulatory endothelial function during the postprandial state in patients with metabolic syndrome: From the LIPGENE study

(2009) Pablo Perez-Martinez et al.   ATHEROSCLEROSIS

n-3 PUFA and lipotoxicity

(2009) Pablo Perez-Martinez et al.   BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS

Oxidative stress and metabolic syndrome

(2009) Christian K. Roberts et al.   LIFE SCIENCES

Production of hydrogen peroxide is responsible for the induction of apoptosis by hydroxytyrosol on HL60 cells

(2009) Roberto Fabiani et al.   MOLECULAR NUTRITION & FOOD RESEARCH

LIPGENE food-exchange model for alteration of dietary fat quantity and quality in free-living participants from eight European countries

(2008) Danielle I. Shaw et al.   BRITISH JOURNAL OF NUTRITION

Oxidative stress markers, C-reactive protein and heat shock protein 70 levels in subjects with metabolic syndrome

(2008) Ferah Armutcu et al.   CLINICAL CHEMISTRY AND LABORATORY MEDICINE

Fat overload aggravates oxidative stress in patients with the metabolic syndrome

(2008) F. Cardona et al.   EUROPEAN JOURNAL OF CLINICAL INVESTIGATION

Methionine in proteins defends against oxidative stress

(2008) Shen Luo et al.   FASEB JOURNAL

Fatty acids as modulators of the cellular production of reactive oxygen species

(2008) Peter Schönfeld et al.   FREE RADICAL BIOLOGY AND MEDICINE

High-fat, energy-dense, fast-food–style breakfast results in an increase in oxidative stress in metabolic syndrome

(2008) Sridevi Devaraj et al.   METABOLISM-CLINICAL AND EXPERIMENTAL

Nrf2 signaling: An adaptive response pathway for protection against environmental toxic insults

(2007) W OSBURN et al.   MUTATION RESEARCH-REVIEWS IN MUTATION RESEARCH