A Role for miR-145 in Pulmonary Arterial Hypertension

Atheroprotective communication between endothelial cells and smooth muscle cells through miRNAs

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MicroRNA and vascular remodelling in acute vascular injury and pulmonary vascular remodelling

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Transforming Growth Factor-β1 (TGF-β1) Utilizes Distinct Pathways for the Transcriptional Activation of MicroRNA 143/145 in Human Coronary Artery Smooth Muscle Cells

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Down-regulation of Krüppel-like Factor-4 (KLF4) by MicroRNA-143/145 Is Critical for Modulation of Vascular Smooth Muscle Cell Phenotype by Transforming Growth Factor-β and Bone Morphogenetic Protein 4

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miR-145 is differentially regulated by TGF-β1 and ischaemia and targets Disabled-2 expression and wnt/β-catenin activity

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Role for miR-204 in human pulmonary arterial hypertension

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Dynamic Changes in Lung MicroRNA Profiles During the Development of Pulmonary Hypertension due to Chronic Hypoxia and Monocrotaline

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miR-145, miR-133a and miR-133b: Tumor-suppressive miRNAs target FSCN1 in esophageal squamous cell carcinoma

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MicroRNA control of podosome formation in vascular smooth muscle cells in vivo and in vitro

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Identification of non-coding RNAs embracing microRNA-143/145 cluster

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Mammalian microRNAs predominantly act to decrease target mRNA levels

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MicroRNAs in Cardiovascular Diseases: Biology and Potential Clinical Applications

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Molecular mechanisms controlling E-cadherin expression in breast cancer

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MicroRNA-145 Regulates OCT4, SOX2, and KLF4 and Represses Pluripotency in Human Embryonic Stem Cells

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MicroRNAs: Target Recognition and Regulatory Functions

(2009) David P. Bartel   CELL

The knockout of miR-143 and -145 alters smooth muscle cell maintenance and vascular homeostasis in mice: correlates with human disease

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MicroRNA-145, a Novel Smooth Muscle Cell Phenotypic Marker and Modulator, Controls Vascular Neointimal Lesion Formation

(2009) Yunhui Cheng et al.   CIRCULATION RESEARCH

MicroRNAs miR-143 and miR-145 modulate cytoskeletal dynamics and responsiveness of smooth muscle cells to injury

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Wnt signaling regulates smooth muscle precursor development in the mouse lung via a tenascin C/PDGFR pathway

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Acquisition of the contractile phenotype by murine arterial smooth muscle cells depends on the Mir143/145 gene cluster

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Dab2 stabilizes Axin and attenuates Wnt/β-catenin signaling by preventing protein phosphatase 1 (PP1)–Axin interactions

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Mice expressing BMPR2R899X transgene in smooth muscle develop pulmonary vascular lesions

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Role of microRNAs in vascular diseases, inflammation, and angiogenesis

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SMAD proteins control DROSHA-mediated microRNA maturation

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