Lipopolysaccharide Regulation of Intestinal Tight Junction Permeability Is Mediated by TLR4 Signal Transduction Pathway Activation of FAK and MyD88
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Title
Lipopolysaccharide Regulation of Intestinal Tight Junction Permeability Is Mediated by TLR4 Signal Transduction Pathway Activation of FAK and MyD88
Authors
Keywords
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Journal
JOURNAL OF IMMUNOLOGY
Volume 195, Issue 10, Pages 4999-5010
Publisher
The American Association of Immunologists
Online
2015-10-15
DOI
10.4049/jimmunol.1402598
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Note: Only part of the references are listed.- Lipopolysaccharide Causes an Increase in Intestinal Tight Junction Permeability in Vitro and in Vivo by Inducing Enterocyte Membrane Expression and Localization of TLR-4 and CD14
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- Probiotic mixture VSL#3 protects the epithelial barrier by maintaining tight junction protein expression and preventing apoptosis in a murine model of colitis
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- Changes in intestinal mucosal immune barrier in rats withendotoxemia
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- Human Endotoxemia as a Model of Systemic Inflammation
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- Protective Effects of Lactoferrin against Intestinal Mucosal Damage Induced by Lipopolysaccharide in Human Intestinal Caco-2 Cells
- (2008) Yoshihiko HIROTANI et al. YAKUGAKU ZASSHI-JOURNAL OF THE PHARMACEUTICAL SOCIETY OF JAPAN
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