Journal
CELL METABOLISM
Volume 20, Issue 6, Pages 937-938Publisher
CELL PRESS
DOI: 10.1016/j.cmet.2014.11.010
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Funding
- NCCIH NIH HHS [R01 AT008623] Funding Source: Medline
- NIDDK NIH HHS [R01 DK073338] Funding Source: Medline
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Obesity and the associated state of subchronic inflammation are risk factors for numerous pathologies, including carcinogenesis. Recently, Schulz et al. (2014) demonstrated that high-fat diet-induced intestinal dysbiosis promotes cancer development in K-ras(G12Dint) mice without inducing obesity or mucosal inflammation, positioning microbial activities as a central component of diet-induced carcinogenesis.
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