Deficiency in Bim, Bid and Bbc3 (Puma) do not prevent axonal injury induced death

Bax regulates primary necrosis through mitochondrial dynamics

(2012) R. S. Whelan et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

The Bcl-2 family member BIM has multiple glaucoma-relevant functions in DBA/2J mice

(2012) Jeffrey M. Harder et al.   Scientific Reports

Can the analysis of BH3-only protein knockout mice clarify the issue of ‘direct versus indirect’ activation of Bax and Bak?

(2011) A Villunger et al.   CELL DEATH AND DIFFERENTIATION

BH3-only Activator Proteins Bid and Bim Are Dispensable for Bak/Bax-dependent Thrombocyte Apoptosis Induced by Bcl-xL Deficiency

(2011) Takahiro Kodama et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

In vivo Contributions of BH3-Only Proteins to Neuronal Death Following Seizures, Ischemia, and Traumatic Brain Injury

(2011) Tobias Engel et al.   JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM

BBC3 (PUMA) regulates developmental apoptosis but not axonal injury induced death in the retina

(2011) Jeffrey M Harder et al.   Molecular Neurodegeneration

BID, BIM, and PUMA Are Essential for Activation of the BAX- and BAK-Dependent Cell Death Program

(2010) D. Ren et al.   SCIENCE

Stepwise Activation of BAX and BAK by tBID, BIM, and PUMA Initiates Mitochondrial Apoptosis

(2009) Hyungjin Kim et al.   MOLECULAR CELL