Intracellular zinc release-activated ERK-dependent GSK-3β–p53 and Noxa–Mcl-1 signaling are both involved in cardiac ischemic-reperfusion injury

Noxa is necessary for hydrogen peroxide-induced caspase-dependent cell death

(2010) Tomonori Aikawa et al.   FEBS LETTERS

The critical role of intracellular zinc in adenosine A2 receptor activation induced cardioprotection against reperfusion injury

(2010) Rachel McIntosh et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

An ERK-dependent pathway to Noxa expression regulates apoptosis by platinum-based chemotherapeutic drugs

(2010) C Sheridan et al.   ONCOGENE

GSK-3β, a Therapeutic Target for Cardiomyocyte Protection

(2009) Tetsuji Miura et al.   CIRCULATION JOURNAL

ERK and cell death: Mechanisms of ERK-induced cell death - apoptosis, autophagy and senescence

(2009) Sebastien Cagnol et al.   FEBS Journal

Inhibition of GSK3β by Postconditioning Is Required to Prevent Opening of the Mitochondrial Permeability Transition Pore During Reperfusion

(2008) Ludovic Gomez et al.   CIRCULATION

Glycogen Synthase Kinase 3 Inhibition Slows Mitochondrial Adenine Nucleotide Transport and Regulates Voltage-Dependent Anion Channel Phosphorylation

(2008) Samarjit Das et al.   CIRCULATION RESEARCH

MAPK regulate p53-dependent cell death induced by benzo[a]pyrene: Involvement of p53 phosphorylation and acetylation

(2008) T. Lin et al.   TOXICOLOGY

Apoptosis: controlled demolition at the cellular level

(2007) Rebecca C. Taylor et al.   NATURE REVIEWS MOLECULAR CELL BIOLOGY