Terminally differentiated astrocytes lack DNA damage response signaling and are radioresistant but retain DNA repair proficiency

Induction and persistence of radiation-induced DNA damage is more pronounced in young animals than in old animals

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ATM is the Predominant Kinase Involved in the Phosphorylation of Histone H2AX after Heating

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53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair

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Replication stress and oxidative damage contribute to aberrant constitutive activation of DNA damage signalling in human gliomas

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Dynamic Dependence on ATR and ATM for Double-Strand Break Repair in Human Embryonic Stem Cells and Neural Descendants

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(2010) M. R. Freeman   SCIENCE

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(2010) William J. Schull   STEM CELLS

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(2009) Brandi L. Mahaney et al.   BIOCHEMICAL JOURNAL

Astrocytes in the damaged brain: Molecular and cellular insights into their reactive response and healing potential

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Molecular Mechanism of Protein Assembly on DNA Double-strand Breaks in the Non-homologous End-joining Pathway

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An optimized experimental strategy for efficient conversion of embryonic stem (ES)-derived mouse neural stem (NS) cells into a nearly homogeneous mature neuronal population

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Tissue-specific DNA-PK-dependent H2AX phosphorylation and γ-H2AX elimination after X-irradiation in vivo

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The role of the DNA damage response in neuronal development, organization and maintenance

(2008) Ari Barzilai et al.   DNA REPAIR

 -H2AX in recognition and signaling of DNA double-strand breaks in the context of chromatin

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Impact of the comet assay in radiobiology

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