γH2AX foci on apparently intact mitotic chromosomes: Not signatures of misrejoining events but signals of unresolved DNA damage

γH2AX foci formation in the absence of DNA damage: Mitotic H2AX phosphorylation is mediated by the DNA-PKcs/CHK2 pathway

(2013) Wen-Zhi Tu et al.   FEBS LETTERS

The Telomere Deprotection Response Is Functionally Distinct from the Genomic DNA Damage Response

(2013) Anthony J. Cesare et al.   MOLECULAR CELL

Genome-wide reorganization of histone H2AX toward particular fragile sites on cell activation

(2013) J. Seo et al.   NUCLEIC ACIDS RESEARCH

Telomere dysfunction induced by chemotherapeutic agents and radiation in normal human cells

(2012) Ping Li et al.   INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY

Eroded human telomeres are more prone to remain uncapped and to trigger a G2 checkpoint response

(2012) Laurent Jullien et al.   NUCLEIC ACIDS RESEARCH

γH2AX foci analysis for monitoring DNA double-strand break repair: Strengths, limitations and optimization

(2011) Markus Löbrich et al.   CELL CYCLE

DNA Damage Response Is Suppressed by the High Cyclin-dependent Kinase 1 Activity in Mitotic Mammalian Cells

(2011) Wei Zhang et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

γH2Ax: Biomarker of Damage or Functional Participant in DNA Repair “All that Glitters Is not Gold!”

(2011) James E. Cleaver   PHOTOCHEMISTRY AND PHOTOBIOLOGY

DNA damage foci in mitosis are devoid of 53BP1

(2010) Glyn Nelson et al.   CELL CYCLE

p53 Prevents Entry into Mitosis with Uncapped Telomeres

(2010) Maria Thanasoula et al.   CURRENT BIOLOGY

DNA damage signaling in response to double-strand breaks during mitosis

(2010) Simona Giunta et al.   JOURNAL OF CELL BIOLOGY

Mammalian Telomeres Resemble Fragile Sites and Require TRF1 for Efficient Replication

(2009) Agnel Sfeir et al.   CELL

Breaksinvisibleto the DNA damage response machinery accumulate in ATM-deficient cells

(2009) Marta Martín et al.   GENES CHROMOSOMES & CANCER

Signatures of DNA double strand breaks produced in irradiated G1 and G2 cells persist into mitosis

(2009) Takamitsu A Kato et al.   JOURNAL OF CELLULAR PHYSIOLOGY

Mechanism of elimination of phosphorylated histone H2AX from chromatin after repair of DNA double-strand breaks

(2009) M.P. Svetlova et al.   MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS

Spontaneous occurrence of telomeric DNA damage response in the absence of chromosome fusions

(2009) Anthony J Cesare et al.   NATURE STRUCTURAL & MOLECULAR BIOLOGY

Mre11 Dimers Coordinate DNA End Bridging and Nuclease Processing in Double-Strand-Break Repair

(2008) R. Scott Williams et al.   CELL

 -H2AX in recognition and signaling of DNA double-strand breaks in the context of chromatin

(2008) A. Kinner et al.   NUCLEIC ACIDS RESEARCH

Comparison of the induction and disappearance of DNA double strand breaks and γ-H2AX foci after irradiation of chromosomes in G1-phase or in condensed metaphase cells

(2007) Takamitsu A. Kato et al.   MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS