A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells

The choice between p53-induced senescence and quiescence is determined in part by the mTOR pathway

(2016) Lioubov G. Korotchkina et al.   Aging-US

Rapamycin decelerates cellular senescence

(2011) Zoya N. Demidenko et al.   CELL CYCLE

AP-1 Is a Component of the Transcriptional Network Regulated by GSK-3 in Quiescent Cells

(2011) John W. Tullai et al.   PLoS One

Regulation of Gene Expression by PI3K in Mouse Growth Plate Chondrocytes

(2010) Veronica Ulici et al.   PLoS One

Identification of Y-Box Binding Protein 1 As a Core Regulator of MEK/ERK Pathway-Dependent Gene Signatures in Colorectal Cancer Cells

(2010) Karsten Jürchott et al.   PLoS Genetics

GSK-3 Represses Growth Factor-inducible Genes by Inhibiting NF-κB in Quiescent Cells

(2009) Julie R. Graham et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Shared Principles in NF-κB Signaling

(2008) Matthew S. Hayden et al.   CELL

Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis

(2008) Frank Götschel et al.   EXPERIMENTAL CELL RESEARCH

Hepatocyte Growth Factor Suppresses Proinflammatory NFκB Activation through GSK3β Inactivation in Renal Tubular Epithelial Cells

(2008) Rujun Gong et al.   JOURNAL OF BIOLOGICAL CHEMISTRY