Extracellular nucleotide inhibits cell proliferation and negatively regulates Toll-like receptor 4 signalling in human progenitor endothelial cells

The expression of functional toll-like receptor 4 is associated with proliferation and maintenance of stem cell phenotype in endothelial progenitor cells (EPCs)

(2010) Jin He et al.   JOURNAL OF CELLULAR BIOCHEMISTRY

ATP-Sensitive K+ Channel-Deficient Dilated Cardiomyopathy Proteome Remodeled by Embryonic Stem Cell Therapy

(2010) Jelena Zlatkovic-Lindor et al.   STEM CELLS

Airway Delivery of Low-Dose Miglustat Normalizes Nasal Potential Difference in F508del Cystic Fibrosis Mice

(2009) Bob Lubamba et al.   AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE

P2Y2 Receptor Transcription Is Increased by NF- B and Stimulates Cyclooxygenase-2 Expression and PGE2 Released by Intestinal Epithelial Cells

(2009) E. Degagne et al.   JOURNAL OF IMMUNOLOGY

A Novel, Highly Selective, Tight Binding I B Kinase-2 (IKK-2) Inhibitor: A Tool to Correlate IKK-2 Activity to the Fate and Functions of the Components of the Nuclear Factor- B Pathway in Arthritis-Relevant Cells and Animal Models

(2009) G. Mbalaviele et al.   JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS

“Host tissue damage” signal ATP impairs IL-12 and IFNγ secretion in LPS stimulated whole human blood

(2008) Marek Nalos et al.   INTENSIVE CARE MEDICINE

Divergent roles of endothelial NF-κB in multiple organ injury and bacterial clearance in mouse models of sepsis

(2008) Xiaobing Ye et al.   JOURNAL OF EXPERIMENTAL MEDICINE

HIF-1α protein is an essential factor for protection of myeloid cells against LPS-induced depletion of ATP and apoptosis that supports Toll-like receptor 4-mediated production of IL-6

(2008) Harjinder Lall et al.   MOLECULAR IMMUNOLOGY

Stem-cell therapy for cardiac disease

(2008) Vincent F. M. Segers et al.   NATURE

ATP is released by monocytes stimulated with pathogen-sensing receptor ligands and induces IL-1  and IL-18 secretion in an autocrine way

(2008) A. Piccini et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA