A review of crosstalk between MAPK and Wnt signals and its impact on cartilage regeneration
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Title
A review of crosstalk between MAPK and Wnt signals and its impact on cartilage regeneration
Authors
Keywords
Chondrogenesis, Mitogen-activated protein kinase (MAPK), MAPK signal, Wnt signal, Stem cell, Osteoarthritis
Journal
CELL AND TISSUE RESEARCH
Volume 358, Issue 3, Pages 633-649
Publisher
Springer Nature
Online
2014-10-13
DOI
10.1007/s00441-014-2010-x
References
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- p38 mitogen-activated protein kinase regulates canonical Wnt- -catenin signaling by inactivation of GSK3
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- G o mediates WNT-JNK signaling through Dishevelled 1 and 3, RhoA family members, and MEKK 1 and 4 in mammalian cells
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- Wnt/β-catenin signaling stimulates matrix catabolic genes and activity in articular chondrocytes: its possible role in joint degeneration
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- Phosphorylation by p38 MAPK as an Alternative Pathway for GSK3 Inactivation
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- Chondrogenic Differentiation of Human Mesenchymal Stem Cells in Three-Dimensional Alginate Gels
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