Substance P induces adverse myocardial remodelling via a mechanism involving cardiac mast cells

Cardiac mast cells: the centrepiece in adverse myocardial remodelling

(2010) S. P. Levick et al.   CARDIOVASCULAR RESEARCH

Differential Cardiac Remodeling in Preload Versus Afterload

(2010) Karl Toischer et al.   CIRCULATION

Sympathetic Nervous System Modulation of Inflammation and Remodeling in the Hypertensive Heart

(2010) Scott P. Levick et al.   HYPERTENSION

Interleukin 6 Mediates Myocardial Fibrosis, Concentric Hypertrophy, and Diastolic Dysfunction in Rats

(2010) Giselle C. Meléndez et al.   HYPERTENSION

Inhibition of matrix metalloproteinase activity prevents increases in myocardial tumor necrosis factor-α

(2010) David B. Murray et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

TNF-α inhibition attenuates adverse myocardial remodeling in a rat model of volume overload

(2009) Lynetta J. Jobe et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Cardiac Mast Cells Mediate Left Ventricular Fibrosis in the Hypertensive Rat Heart

(2009) Scott P. Levick et al.   HYPERTENSION

A novel technique for isolating functional mast cells from the heart

(2008) L. G. Morgan et al.   INFLAMMATION RESEARCH

Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient rats

(2008) Scott P. Levick et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Substance P regulates the expression of matrix metalloproteinases and tissue inhibitors of metalloproteinase in cultured human gingival fibroblasts

(2008) P. R. Cury et al.   JOURNAL OF PERIODONTAL RESEARCH