Mutations in sodium channel β-subunit SCN3B are associated with early-onset lone atrial fibrillation
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Title
Mutations in sodium channel β-subunit SCN3B are associated with early-onset lone atrial fibrillation
Authors
Keywords
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Journal
CARDIOVASCULAR RESEARCH
Volume 89, Issue 4, Pages 786-793
Publisher
Oxford University Press (OUP)
Online
2010-11-05
DOI
10.1093/cvr/cvq348
References
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Related references
Note: Only part of the references are listed.- Scn3b knockout mice exhibit abnormal sino-atrial and cardiac conduction properties
- (2009) P. Hakim et al. Acta Physiologica
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- (2009) Dan Hu et al. Circulation-Cardiovascular Genetics
- Genetics of familial atrial fibrillation
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- New-onset atrial fibrillation as first clinical manifestation of latent Brugada syndrome: prevalence and clinical significance
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- (2009) Ewa Soltysinska et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Scn3b knockout mice exhibit abnormal ventricular electrophysiological properties
- (2009) Parvez Hakim et al. PROGRESS IN BIOPHYSICS & MOLECULAR BIOLOGY
- Analyses of a novel SCN5A mutation (C1850S): conduction vs. repolarization disorder hypotheses in the Brugada syndrome
- (2008) Séverine Petitprez et al. CARDIOVASCULAR RESEARCH
- Cardiac Sodium Channel ( SCN5A ) Variants Associated with Atrial Fibrillation
- (2008) Dawood Darbar et al. CIRCULATION
- Cardiac sodium channel mutation in atrial fibrillation
- (2007) Patrick T. Ellinor et al. HEART RHYTHM
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